about
Loss of Nek11 Prevents G2/M Arrest and Promotes Cell Death in HCT116 Colorectal Cancer Cells Exposed to Therapeutic DNA Damaging AgentsStra6, a retinoic acid-responsive gene, participates in p53-induced apoptosis after DNA damage.Retinal pigment epithelial cell DNA is damaged by exposure to benzo[a]pyrene, a constituent of cigarette smoke.Improving and predicting radiosensitivity in muscle invasive bladder cancer.Comet assay measures of DNA damage as biomarkers of irinotecan response in colorectal cancer in vitro and in vivoInduction of strand breaks in polyribonucleotides and DNA by the sulphate radical anion: role of electron loss centres as precursors of strand breakage.Fractionated radiation exposure amplifies the radioresistant nature of prostate cancer cells.SIP1 protein protects cells from DNA damage-induced apoptosis and has independent prognostic value in bladder cancer.Simplified method for the collection, storage, and comet assay analysis of DNA damage in whole blood.Radiation-induced transgenerational alterations in genome stability and DNA damage.Measurements using the alkaline comet assay predict bladder cancer cell radiosensitivity.Anthocyans as tertiary chemopreventive agents in bladder cancer: anti-oxidant mechanisms and interaction with mitomycin C.Comet assay measures of DNA damage are predictive of bladder cancer cell treatment sensitivity in vitro and outcome in vivo.DNA-repair measurements by use of the modified comet assay: an inter-laboratory comparison within the European Comet Assay Validation Group (ECVAG).Speciation of oxaliplatin adducts with DNA nucleotides.Inter-laboratory variation in DNA damage using a standard comet assay protocol.DNA strand breaks and hypoxia response inhibition mediate the radiosensitisation effect of nitric oxide donors on prostate cancer under varying oxygen conditions.Determination of cisplatin 1,2-intrastrand guanine-guanine DNA adducts in human leukocytes by high-performance liquid chromatography coupled to inductively coupled plasma mass spectrometry.Introducing a true internal standard for the Comet assay to minimize intra- and inter-experiment variability in measures of DNA damage and repair.Cytotoxicity and gene expression profiling of two hydroxylated polybrominated diphenyl ethers in human H295R adrenocortical carcinoma cells.Multiple end-point analysis reveals cisplatin damage tolerance to be a chemoresistance mechanism in a NSCLC model: implications for predictive testing.Extended treatment with physiologic concentrations of dietary phytochemicals results in altered gene expression, reduced growth, and apoptosis of cancer cells.Investigation of the role of extracellular H2O2 and transition metal ions in the genotoxic action of ascorbic acid in cell culture models.Phosphate alkylation in different DNA substrates: the role of local DNA sequence and electrophile character in determining the nonrandom nature of phosphotriester adduct formation.Detection of oxaliplatin-induced DNA crosslinks in vitro and in cancer patients using the alkaline comet assay.Azinomycin inspired bisepoxides: influence of linker structure on in vitro cytotoxicity and DNA interstrand cross-linking.BTK Modulates p53 Activity to Enhance Apoptotic and Senescent Responses.Effects of postirradiation temperature on the yields of radiation-induced single- and double-strand breakage in SV40 DNA.DNMTs are required for delayed genome instability caused by radiation.HDAC inhibitor confers radiosensitivity to prostate stem-like cellsPosttranscriptional Upregulation of p53 by Reactive Oxygen Species in Chronic Lymphocytic Leukemia.32P-postlabelling approaches for the detection of 8-oxo-2'-deoxyguanosine-3'-monophosphate in DNA.Characterization of DNA damage at purine residues in oligonucleotides and calf thymus DNA induced by the mutagen 1-nitrosoindole-3-acetonitrile.Deoxycytidine glyoxal: lesion induction and evidence of repair following vitamin C supplementation in vivo.Yield of strand breaks as a function of scavenger concentration and LET for SV40 irradiated with 4He ions.Establishment of the National Cancer Research Institute Clinical and Translational Radiotherapy Research Working Group (CTRad) Biomarker Support Network.Structure and mobility of electron gain and loss centres in proteins.Variation of DNA damage levels in peripheral blood mononuclear cells isolated in different laboratories.Variation in the measurement of DNA damage by comet assay measured by the ECVAG inter-laboratory validation trial.Potential clinical exploitation of the radiation-induced DNA damage response.
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P50
description
onderzoeker
@nl
researcher
@en
հետազոտող
@hy
name
George D Jones
@ast
George D Jones
@en
George D Jones
@es
George D Jones
@nl
جورج دي جونز
@ar
type
label
George D Jones
@ast
George D Jones
@en
George D Jones
@es
George D Jones
@nl
جورج دي جونز
@ar
prefLabel
George D Jones
@ast
George D Jones
@en
George D Jones
@es
George D Jones
@nl
جورج دي جونز
@ar
P106
P1153
7406141746
P31
P496
0000-0002-9543-2206