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Cyclosporine A induces apoptotic and autophagic cell death in rat pituitary GH3 cellsHistone methylation levels correlate with TGFBIp and extracellular matrix gene expression in normal and granular corneal dystrophy type 2 corneal fibroblasts.Lithium inhibits tumor lymphangiogenesis and metastasis through the inhibition of TGFBIp expression in cancer cellsDecreased catalase expression and increased susceptibility to oxidative stress in primary cultured corneal fibroblasts from patients with granular corneal dystrophy type II.Role of TGFBIp in Wound Healing and Mucin Expression in Corneal Epithelial Cells.Molecular Pathogenesis of Corneal Dystrophies: Schnyder Dystrophy and Granular Corneal Dystrophy type 2.Autophagy in granular corneal dystrophy type 2.Pathogenesis and treatments of TGFBI corneal dystrophies.TGF-β regulates TGFBIp expression in corneal fibroblasts via miR-21, miR-181a, and Smad signaling.Melatonin prevents nitric oxide-induced apoptosis by increasing the interaction between 14-3-3beta and p-Bad in SK-N-MC cells.Adult-Onset Vitelliform Macular Dystrophy caused by BEST1 p.Ile38Ser Mutation is a Mild Form of Best Vitelliform Macular DystrophyDisrupted cell cycle arrest and reduced proliferation in corneal fibroblasts from GCD2 patients: a potential role for altered autophagy flux.Melatonin induces autophagy via an mTOR-dependent pathway and enhances clearance of mutant-TGFBIp.Inhibitory Effect of Tranilast on Transforming Growth Factor-Beta-Induced Protein in Granular Corneal Dystrophy Type 2 Corneal Fibroblasts.Inhibition of TGFBIp expression reduces lymphangiogenesis and tumor metastasis.Melatonin reduces endoplasmic reticulum stress and corneal dystrophy-associated TGFBIp through activation of endoplasmic reticulum-associated protein degradation.A Novel BEST1 Mutation in Autosomal Recessive Bestrophinopathy.4-Phenylbutyric acid reduces mutant-TGFBIp levels and ER stress through activation of ERAD pathway in corneal fibroblasts of granular corneal dystrophy type 2.Inhibition of TGFBIp expression by lithium: implications for TGFBI-linked corneal dystrophy therapy.Autophagy is induced by raptor degradation via the ubiquitin/proteasome system in granular corneal dystrophy type 2.Toll-like receptor 4 initiates an innate immune response to lipopolysaccharide in human conjunctival epithelial cellsMelatonin protects against oxidative stress in granular corneal dystrophy type 2 corneal fibroblasts by mechanisms that involve membrane melatonin receptorsAPP processing and metabolism in corneal fibroblasts and epithelium as a potential biomarker for Alzheimer's disease
P50
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P50
description
researcher
@en
wetenschapper
@nl
հետազոտող
@hy
name
Seung-Il Choi
@ast
Seung-Il Choi
@en
Seung-Il Choi
@es
Seung-Il Choi
@nl
type
label
Seung-Il Choi
@ast
Seung-Il Choi
@en
Seung-Il Choi
@es
Seung-Il Choi
@nl
prefLabel
Seung-Il Choi
@ast
Seung-Il Choi
@en
Seung-Il Choi
@es
Seung-Il Choi
@nl
P106
P31
P496
0000-0001-7168-8795