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Increased Na⁺/Ca²⁺ exchanger expression/activity constitutes a point of inflection in the progression to heart failure of hypertensive ratsUltradeformable Archaeosomes for Needle Free Nanovaccination with Leishmania braziliensis AntigensDihydropyridine-sensitive skeletal muscle Ca channels in polarized planar bilayers. 1. Kinetics and voltage dependence of gating.Dihydropyridine-sensitive skeletal muscle Ca channels in polarized planar bilayers. 2. Effects of phosphorylation by cAMP-dependent protein kinaseSelective cytotoxicity of PAMAM G5 core--PAMAM G2.5 shell tecto-dendrimers on melanoma cells.Calcium-calmodulin dependent protein kinase II (CaMKII): a main signal responsible for early reperfusion arrhythmias.Ca2+/calmodulin kinase II increases ryanodine binding and Ca2+-induced sarcoplasmic reticulum Ca2+ release kinetics during beta-adrenergic stimulation.Role of phospholamban phosphorylation on Thr17 in cardiac physiological and pathological conditions.The importance of the Thr17 residue of phospholamban as a phosphorylation site under physiological and pathological conditions.Ca2+/calmodulin-dependent protein kinase: a key component in the contractile recovery from acidosis.Increased intracellular Ca2+ and SR Ca2+ load contribute to arrhythmias after acidosis in rat heart. Role of Ca2+/calmodulin-dependent protein kinase II.Physiologic and pharmacologic factors that affect myocardial relaxation.Ca(2+)/calmodulin-dependent protein kinase II contributes to intracellular pH recovery from acidosis via Na(+)/H(+) exchanger activation.Reversible redox modifications of ryanodine receptor ameliorate ventricular arrhythmias in the ischemic-reperfused heart.Phosphorylation of phospholamban in the intact heart. A study on the physiological role of the Ca(2+)-calmodulin-dependent protein kinase system.Phospholamban phosphorylation in ischemia-reperfused heart. Effect of pacing during ischemia and response to a beta-adrenergic challenge.Phospholamban phosphorylation sites enhance the recovery of intracellular Ca2+ after perfusion arrest in isolated, perfused mouse heart.Time course and mechanisms of phosphorylation of phospholamban residues in ischemia-reperfused rat hearts. Dissociation of phospholamban phosphorylation pathways.The relative relevance of phosphorylation of the Thr(17) residue of phospholamban is different at different levels of beta-adrenergic stimulation.Role of dual-site phospholamban phosphorylation in the stunned heart: insights from phospholamban site-specific mutants.Phosphorylation of phospholamban in ischemia-reperfusion injury: functional role of Thr17 residue.Frequency-dependent acceleration of relaxation in mammalian heart: a property not relying on phospholamban and SERCA2a phosphorylation.Role of phosphorylation of Thr(17) residue of phospholamban in mechanical recovery during hypercapnic acidosis.Ca2+-calmodulin-dependent protein kinase phosphorylation of ryanodine receptor may contribute to the beta-adrenergic regulation of myocardial contractility independently of increases in heart rate.Characteristics of ryanodine-induced tetani in the perfused rat heart. Tetanic tension is not the highest force that cardiac muscle can generate.Early effects of Epac depend on the fine-tuning of the sarcoplasmic reticulum Ca2+ handling in cardiomyocytes.Role of CaMKII in post acidosis arrhythmias: a simulation study using a human myocyte model.Subchronic microcystin-LR exposure increased hepatic apoptosis and induced compensatory mechanisms in mice.
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P50
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Argentijns onderzoekster
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Investigadora
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name
Cecilia Beatriz Mundiña
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Cecilia Beatriz Mundiña
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Cecilia Beatriz Mundiña
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Cecilia Beatriz Mundiña
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Cecilia Beatriz Mundiña
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Cecilia Beatriz Mundiña
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Cecilia Beatriz Mundiña
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Cecilia Beatriz Mundiña
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Cecilia Beatriz Mundiña
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Cecilia Beatriz Mundiña
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Cecilia Beatriz Mundiña
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Cecilia Beatriz Mundiña
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C Mundina
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Cecilia Beatriz Mundiña
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Cecilia Beatriz Mundiña
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Cecilia Beatriz Mundiña
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Cecilia Beatriz Mundiña
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Cecilia Beatriz Mundiña
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Cecilia Beatriz Mundiña
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Cecilia Beatriz Mundiña
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Cecilia Beatriz Mundiña-Weilenmann
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