about
Distinct APC subtypes drive spatially segregated CD4+ and CD8+ T-cell effector activity during skin infection with HSV-1Strikingly poor CD8+ T-cell immunogenicity of vaccinia virus strain MVA in BALB/c mice.Lytic gene expression is frequent in HSV-1 latent infection and correlates with the engagement of a cell-intrinsic transcriptional responseAnalysis of A47, an immunoprevalent protein of vaccinia virus, leads to a reevaluation of the total antiviral CD8+ T cell response.Lytic Promoters Express Protein during Herpes Simplex Virus Latency.Engineering herpes simplex viruses by infection-transfection methods including recombination site targeting by CRISPR/Cas9 nucleases.Systems-guided forward genetic screen reveals a critical role of the replication stress response protein ETAA1 in T cell clonal expansion.Local proliferation maintains a stable pool of tissue-resident memory T cells after antiviral recall responses.Effective Priming of Herpes Simplex Virus-Specific CD8+ T Cells In Vivo Does Not Require Infected Dendritic Cells.Increasing antigen presentation on HSV-1-infected cells increases lesion size but does not alter neural infection or latency.Qualitative Differences in Capsidless L-Particles Released as a By-Product of Bovine Herpesvirus 1 and Herpes Simplex Virus 1 InfectionsFunctional paralysis of human natural killer cells by alphaherpesviruses.
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description
researcher
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wetenschapper
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հետազոտող
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name
Tiffany A Russell
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Tiffany A Russell
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Tiffany A Russell
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Tiffany A Russell
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Tiffany A Russell
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Tiffany A Russell
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Tiffany A Russell
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type
label
Tiffany A Russell
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Tiffany A Russell
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Tiffany A Russell
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Tiffany A Russell
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Tiffany A Russell
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Tiffany A Russell
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Tiffany A Russell
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prefLabel
Tiffany A Russell
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Tiffany A Russell
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Tiffany A Russell
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Tiffany A Russell
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Tiffany A Russell
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Tiffany A Russell
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Tiffany A Russell
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P106
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0000-0003-0188-9393