about
Tight junction defects in patients with atopic dermatitis.Toll-like receptor 3 activation is required for normal skin barrier repair following UV damage.Phosphatase activity of histidine kinase EnvZ without kinase catalytic domain.NBK/BIK antagonizes MCL-1 and BCL-XL and activates BAK-mediated apoptosis in response to protein synthesis inhibition.The cutaneous innate immune response in patients with atopic dermatitis.Epidermal tight junctions in health and disease.Characterization of V. cholerae T3SS-dependent cytotoxicity in cultured intestinal epithelial cells.Human organic anion transporter hOAT1 forms homooligomers.Structural and functional studies of the HAMP domain of EnvZ, an osmosensing transmembrane histidine kinase in Escherichia coli.The design and development of Tar-EnvZ chimeric receptors.Atopic dermatitis Subjects Colonized with Staphylococcus aureus have a Distinct Phenotype and Endotype.Functional and structural characterization of EnvZ, an osmosensing histidine kinase of E. coli.Transcription regulation of ompF and ompC by a single transcription factor, OmpR.The critical role of the conserved Thr247 residue in the functioning of the osmosensor EnvZ, a histidine Kinase/Phosphatase, in Escherichia coliMechanism of stabilization of a bacterial collagen triple helix in the absence of hydroxyprolineAntagonistic Effects of IL-4 on IL-17A-Mediated Enhancement of Epidermal Tight Junction Function
P50
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P50
description
researcher ORCID ID=0000-0003-0133-5067
@en
name
Takeshi Yoshida
@ast
Takeshi Yoshida
@en
Takeshi Yoshida
@nl
type
label
Takeshi Yoshida
@ast
Takeshi Yoshida
@en
Takeshi Yoshida
@nl
prefLabel
Takeshi Yoshida
@ast
Takeshi Yoshida
@en
Takeshi Yoshida
@nl
P106
P31
P496
0000-0003-0133-5067