about
A window into the heterogeneity of human cerebrospinal fluid Aβ peptidesCCL8/MCP-2 association analysis in patients with Alzheimer's disease and frontotemporal lobar degenerationPossible association of mitochondrial transcription factor A (TFAM) genotype with sporadic Alzheimer diseaseDetection of the presenilin 1 COOH-terminal fragment in the extracellular compartment: a release enhanced by apoptosisCystatin C is released in association with exosomes: a new tool of neuronal communication which is unbalanced in Alzheimer's disease.Behavioral and neurophysiological effects of transdermal rotigotine in atypical parkinsonism.Rotigotine is safe and efficacious in Atypical Parkinsonism Syndromes induced by both α-synucleinopathy and tauopathy.Comparison of the effects of transdermal and oral rivastigmine on cognitive function and EEG markers in patients with Alzheimer's disease.High prevalence of pathogenic mutations in patients with early-onset dementia detected by sequence analyses of four different genesH1 haplotype of the MAPT gene is associated with lower regional gray matter volume in healthy carriers.Circulating levels of soluble receptor for advanced glycation end products in Alzheimer disease and vascular dementia.TMEM106B regulates progranulin levels and the penetrance of FTLD in GRN mutation carriers.No association between DCP1 genotype and late-onset Alzheimer disease.Value of serum nonceruloplasmin copper for prediction of mild cognitive impairment conversion to Alzheimer disease.Temporal lobe asymmetry in patients with Alzheimer's disease with delusionsSpecific EEG changes associated with atrophy of hippocampus in subjects with mild cognitive impairment and Alzheimer's disease.Medial temporal atrophy but not memory deficit predicts progression to dementia in patients with mild cognitive impairment.EEG upper/low alpha frequency power ratio relates to temporo-parietal brain atrophy and memory performances in mild cognitive impairment.Mapping the effect of APOE epsilon4 on gray matter loss in Alzheimer's disease in vivoIncrease of theta frequency is associated with reduction in regional cerebral blood flow only in subjects with mild cognitive impairment with higher upper alpha/low alpha EEG frequency power ratio.Cerebrospinal fluid biomarkers for Alzheimer's disease: the present and the future.Losing protein in the brain: the case of progranulin.Action and object naming in frontotemporal dementia, progressive supranuclear palsy, and corticobasal degeneration.Semantic memory in Alzheimer's disease: an analysis of category fluency.Progranulin Mutations Affects Brain Oscillatory Activity in Fronto-Temporal Dementia.Effects of Multiple Genetic Loci on Age at Onset in Frontotemporal Dementia.Preliminary evidence of validity of the revised criteria for Alzheimer disease diagnosis: report of 2 cases.Low plasma progranulin levels predict progranulin mutations in frontotemporal lobar degeneration.Blockade of the tumor necrosis factor-related apoptosis inducing ligand death receptor DR5 prevents beta-amyloid neurotoxicity.Apolipoprotein E and alpha brain rhythms in mild cognitive impairment: a multicentric electroencephalogram study.Interaction between tau and alpha-synuclein proteins is impaired in the presence of P301L tau mutation.Reduction of Ca2+ stores and capacitative Ca2+ entry is associated with the familial Alzheimer's disease presenilin-2 T122R mutation and anticipates the onset of dementia.An electronic memory aid to support prospective memory in patients in the early stages of Alzheimer's disease: a pilot study.Genetic Counseling and Testing for Alzheimer's Disease and Frontotemporal Lobar Degeneration: An Italian Consensus Protocol.Risk of mortality and institutionalization in demented patients with delusions.Defective protein kinase C alpha leads to impaired secretion of soluble beta-amyloid precursor protein from Alzheimer's disease fibroblasts.Effect of energy shortage and oxidative stress on amyloid precursor protein metabolism in COS cells.Pharmacogenomics in Alzheimer's disease: a genome-wide association study of response to cholinesterase inhibitors.BACE-2 is overexpressed in Down's syndrome.Inhibition of energy metabolism down-regulates the Alzheimer related presenilin 2 gene.
P50
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P50
description
investigador
@es
researcher
@en
wetenschapper
@nl
name
Giuliano Binetti
@ast
Giuliano Binetti
@en
Giuliano Binetti
@es
Giuliano Binetti
@nl
type
label
Giuliano Binetti
@ast
Giuliano Binetti
@en
Giuliano Binetti
@es
Giuliano Binetti
@nl
prefLabel
Giuliano Binetti
@ast
Giuliano Binetti
@en
Giuliano Binetti
@es
Giuliano Binetti
@nl
P1053
K-4519-2016
P106
P1153
35429391100
P31
P496
0000-0003-2759-5844