about
Effect of simvastatin on cognitive functioning in children with neurofibromatosis type 1: a randomized controlled trialTreatment of neurodevelopmental disorders in adulthoodArc expression identifies the lateral amygdala fear memory trace.The SH3 domain of the Saccharomyces cerevisiae peroxisomal membrane protein Pex13p functions as a docking site for Pex5p, a mobile receptor for the import PTS1-containing proteins.Overexpression of Pex15p, a phosphorylated peroxisomal integral membrane protein required for peroxisome assembly in S.cerevisiae, causes proliferation of the endoplasmic reticulum membraneCytosolic aspartate aminotransferase encoded by the AAT2 gene is targeted to the peroxisomes in oleate-grown Saccharomyces cerevisiae.α-Calcium calmodulin kinase II modulates the temporal structure of hippocampal bursting patternsIn vivo synaptic transmission and morphology in mouse models of Tuberous sclerosis, Fragile X syndrome, Neurofibromatosis type 1, and Costello syndromeCognitive deficits in Tsc1+/- mice in the absence of cerebral lesions and seizures.Spred1 is required for synaptic plasticity and hippocampus-dependent learning.Molecular mechanisms of synaptic plasticity and memory.Motor deficits in neurofibromatosis type 1 mice: the role of the cerebellum.Intrinsic plasticity complements long-term potentiation in parallel fiber input gain control in cerebellar Purkinje cells.Mammalian target of rapamycin complex I (mTORC1) activity in ras homologue enriched in brain (Rheb)-deficient mouse embryonic fibroblastsRheb is essential for murine developmentTemporal and region-specific requirements of αCaMKII in spatial and contextual learning.Ube3a reinstatement identifies distinct developmental windows in a murine Angelman syndrome model.Mouse genetic approaches to investigating calcium/calmodulin-dependent protein kinase II function in plasticity and cognition.A mobile PTS2 receptor for peroxisomal protein import in Pichia pastoris.Behavioral and cognitive outcomes for clinical trials in children with neurofibromatosis type 1alpha-Isoform of calcium-calmodulin-dependent protein kinase II and postsynaptic density protein 95 differentially regulate synaptic expression of NR2A- and NR2B-containing N-methyl-d-aspartate receptors in hippocampus.PAS10 is a tetratricopeptide-repeat protein that is essential for the import of most matrix proteins into peroxisomes of Saccharomyces cerevisiaeGABAergic Neuron-Specific Loss of Ube3a Causes Angelman Syndrome-Like EEG Abnormalities and Enhances Seizure Susceptibility.The molecular, temporal and region-specific requirements of the beta isoform of Calcium/Calmodulin-dependent protein kinase type 2 (CAMK2B) in mouse locomotion.Calcium threshold shift enables frequency-independent control of plasticity by an instructive signalTreatment of Cognitive Deficits in Genetic Disorders: A Systematic Review of Clinical Trials of Diet and Drug Treatments.Interaction of SH-SY5Y cells with nanogratings during neuronal differentiation: comparison with primary neurons.Interdependence of clinical factors predicting cognition in children with tuberous sclerosis complexKinase activity is not required for alphaCaMKII-dependent presynaptic plasticity at CA3-CA1 synapses.Peroxisomal and mitochondrial carnitine acetyltransferases of Saccharomyces cerevisiae are encoded by a single gene.Transport of proteins and metabolites across the impermeable membrane of peroxisomes.Proteins involved in peroxisome biogenesis and functioning.HCN channels are a novel therapeutic target for cognitive dysfunction in Neurofibromatosis type 1.Ca(2+)/calmodulin-dependent protein kinase IIα (αCaMKII) controls the activity of the dopamine transporter: implications for Angelman syndrome.Mechanisms underlying cognitive deficits in a mouse model for Costello Syndrome are distinct from other RASopathy mouse modelsVariation in a range of mTOR-related genes associates with intracranial volume and intellectual disability.Hippocampal synaptic metaplasticity requires inhibitory autophosphorylation of Ca2+/calmodulin-dependent kinase II.Synaptic transmission and plasticity at inputs to murine cerebellar Purkinje cells are largely dispensable for standard nonmotor tasks.Analysis of Cx36 knockout does not support tenet that olivary gap junctions are required for complex spike synchronization and normal motor performance.Derangements of hippocampal calcium/calmodulin-dependent protein kinase II in a mouse model for Angelman mental retardation syndrome.
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0000-0002-3758-1297