about
Pho4 mediates phosphate acquisition in Candida albicans and is vital for stress resistance and metal homeostasisElevated catalase expression in a fungal pathogen is a double-edged sword of iron.Integrative Model of Oxidative Stress Adaptation in the Fungal Pathogen Candida albicans.Blocking two-component signalling enhances Candida albicans virulence and reveals adaptive mechanisms that counteract sustained SAPK activationSfp1 and Rtg3 reciprocally modulate carbon source-conditional stress adaptation in the pathogenic yeast Candida albicansMechanisms Underlying the Delayed Activation of the Cap1 Transcription Factor in Candida albicans following Combinatorial Oxidative and Cationic Stress Important for Phagocytic Potency.The lack of upstream elements of the Cek1 and Hog1 mediated pathways leads to a synthetic lethal phenotype upon osmotic stress in Candida albicans.The transmembrane protein Opy2 mediates activation of the Cek1 MAP kinase in Candida albicans.Stress-induced nuclear accumulation is dispensable for Hog1-dependent gene expression and virulence in a fungal pathogen.Redox Regulation, Rather than Stress-Induced Phosphorylation, of a Hog1 Mitogen-Activated Protein Kinase Modulates Its Nitrosative-Stress-Specific Outputs.
P50
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P50
description
Forscher
@de
chercheur
@fr
investigador
@es
researcher
@en
ricercatore
@it
wetenschapper
@nl
研究者
@zh
name
Carmen M. Herrero-de-Dios
@en
Herrero-de-Dios CM
@ast
Herrero-de-Dios CM
@es
Herrero-de-Dios CM
@nl
type
label
Carmen M. Herrero-de-Dios
@en
Herrero-de-Dios CM
@ast
Herrero-de-Dios CM
@es
Herrero-de-Dios CM
@nl
prefLabel
Carmen M. Herrero-de-Dios
@en
Herrero-de-Dios CM
@ast
Herrero-de-Dios CM
@es
Herrero-de-Dios CM
@nl
P1053
N-4724-2018
P106
P31
P496
0000-0002-2042-8480