about
Enhancing effects of adjuvanted 2009 pandemic H1N1 influenza A vaccine on memory B-cell responses in HIV-infected individualsImpact of HIV on cell survival and antiviral activity of plasmacytoid dendritic cellsT-bet+ B cells are induced by human viral infections and dominate the HIV gp140 response.Nef, macrophages and B cells: a highway for evasionCXCR4/IgG-expressing plasma cells are associated with human gastrointestinal tissue inflammation.Glycosylation, hypogammaglobulinemia, and resistance to viral infectionsAbnormal B cell memory subsets dominate HIV-specific responses in infected individuals.Global analyses of human immune variation reveal baseline predictors of postvaccination responses.HIV-infected individuals receiving effective antiviral therapy for extended periods of time continually replenish their viral reservoir.Congenital B cell lymphocytosis explained by novel germline CARD11 mutations.Human immunodeficiency virus type 1 bound to B cells: relationship to virus replicating in CD4+ T cells and circulating in plasma.Appearance of immature/transitional B cells in HIV-infected individuals with advanced disease: correlation with increased IL-7.B cells in early and chronic HIV infection: evidence for preservation of immune function associated with early initiation of antiretroviral therapy.Broadly neutralizing antibodies suppress HIV in the persistent viral reservoirPersistence of HIV in gut-associated lymphoid tissue despite long-term antiretroviral therapy.B cells in HIV infection and diseaseGene expression and viral prodution in latently infected, resting CD4+ T cells in viremic versus aviremic HIV-infected individuals.Relationship between residual plasma viremia and the size of HIV proviral DNA reservoirs in infected individuals receiving effective antiretroviral therapyPerturbations in B cell responsiveness to CD4+ T cell help in HIV-infected individualsAttenuation of HIV-associated human B cell exhaustion by siRNA downregulation of inhibitory receptorsRebound of plasma viremia following cessation of antiretroviral therapy despite profoundly low levels of HIV reservoir: implications for eradication.Bone marrow plasma cells are a primary source of serum HIV-1-specific antibodies in chronically infected individualsTwo overrepresented B cell populations in HIV-infected individuals undergo apoptosis by different mechanisms.Cold urticaria, immunodeficiency, and autoimmunity related to PLCG2 deletions.Paucity of HIV DNA methylation in latently infected, resting CD4+ T cells from infected individuals receiving antiretroviral therapyB cell homeostasis in chronic hepatitis C virus-related mixed cryoglobulinemia is maintained through naïve B cell apoptosis.A hypermorphic missense mutation in PLCG2, encoding phospholipase Cγ2, causes a dominantly inherited autoinflammatory disease with immunodeficiency.Effect of histone deacetylase inhibitors on HIV production in latently infected, resting CD4(+) T cells from infected individuals receiving effective antiretroviral therapyHumans with chronic granulomatous disease maintain humoral immunologic memory despite low frequencies of circulating memory B cells.Decreased survival of B cells of HIV-viremic patients mediated by altered expression of receptors of the TNF superfamily.Relationship between the frequency of HIV-specific CD8+ T cells and the level of CD38+CD8+ T cells in untreated HIV-infected individualsLoss-of-function mutations in the IL-21 receptor gene cause a primary immunodeficiency syndromeCharacterization of plasmablasts in the blood of HIV-infected viremic individuals: evidence for nonspecific immune activation.Evidence for HIV-associated B cell exhaustion in a dysfunctional memory B cell compartment in HIV-infected viremic individuals.Maturational characteristics of HIV-specific antibodies in viremic individualsCpG oligonucleotides enhance proliferative and effector responses of B Cells in HIV-infected individuals.Pathogenic mechanisms of B-lymphocyte dysfunction in HIV diseaseEffect of antiretroviral therapy on HIV reservoirs in elite controllers.Peripheral blood B cell subset skewing is associated with altered cell cycling and intrinsic resistance to apoptosis and reflects a state of immune activation in chronic hepatitis C virus infectionIL-7 administration drives T cell-cycle entry and expansion in HIV-1 infection
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description
researcher ORCID ID=0000-0002-0163-6911
@en
wetenschapper
@nl
name
Susan Moir
@ast
Susan Moir
@en
Susan Moir
@nl
type
label
Susan Moir
@ast
Susan Moir
@en
Susan Moir
@nl
prefLabel
Susan Moir
@ast
Susan Moir
@en
Susan Moir
@nl
P106
P31
P496
0000-0002-0163-6911