about
Kynurenine is an endothelium-derived relaxing factor produced during inflammationCommon Statistical Pitfalls in Basic Science ResearchPathophysiological role of oxidative stress in systolic and diastolic heart failure and its therapeutic implicationsExpression of 5-lipoxygenase in pulmonary artery endothelial cellsNitroglycerin is preferable to diltiazem for prevention of coronary bypass conduit spasm.Endoscopic versus conventional radial artery harvest for coronary artery bypass grafting: functional and histologic assessment of the conduit.NO forms an adduct with serum albumin that has endothelium-derived relaxing factor-like properties.p38 mitogen-activated protein kinase activates eNOS in endothelial cells by an estrogen receptor alpha-dependent pathway in response to black tea polyphenols.Low-dose alpha-tocopherol improves and high-dose alpha-tocopherol worsens endothelial vasodilator function in cholesterol-fed rabbits.In vivo transfer of nitric oxide between a plasma protein-bound reservoir and low molecular weight thiols.Role of nitric oxide in parasympathetic modulation of beta-adrenergic myocardial contractility in normal dogsEffects of Concord grape juice on ambulatory blood pressure in prehypertension and stage 1 hypertension.Endothelial AMPK activation induces mitochondrial biogenesis and stress adaptation via eNOS-dependent mTORC1 signaling.Plasma microRNAs are associated with atrial fibrillation and change after catheter ablation (the miRhythm study).Vascular oxidative stress and antioxidant protection in atherosclerosis: what do the clinical trials say?Relationship among circulating inflammatory proteins, platelet gene expression, and cardiovascular riskChronic activation of AMP-activated protein kinase prevents 20-hydroxyeicosatetraenoic acid-induced endothelial dysfunction.Measurements of redox control of nitric oxide bioavailability.Oxidative stress and endothelial nitric oxide bioactivity.Reactive oxygen species in cardiovascular disease.Beyond LDL oxidation: ROS in vascular signal transduction.Nutrient sensing by the mitochondrial transcription machinery dictates oxidative phosphorylation.Oxidative stress, antioxidants, and endothelial function.New insights on oxidative stress in the artery wall.Endothelial NADPH oxidase 4 protects ApoE-/- mice from atherosclerotic lesionsDietary antioxidants preserve endothelium-dependent vessel relaxation in cholesterol-fed rabbits.Coronary artery perforation during excimer laser coronary angioplasty. The percutaneous Excimer Laser Coronary Angioplasty Registry.Effect of sulfasalazine on inflammation and endothelial function in patients with established coronary artery disease.The molecular basis of the genesis of basal tone in internal anal sphincter.Predictive value of reactive hyperemia for cardiovascular events in patients with peripheral arterial disease undergoing vascular surgerySuppression of ischemia in arterial occlusive disease by JNK-promoted native collateral artery development.Serum myeloperoxidase levels independently predict endothelial dysfunction in humans.Suppression of the JNK pathway by induction of a metabolic stress response prevents vascular injury and dysfunction.L-2-Oxothiazolidine-4-carboxylic acid reverses endothelial dysfunction in patients with coronary artery disease.α1AMP-activated protein kinase mediates vascular protective effects of exercise.Relation of smoking status to a panel of inflammatory markers: the framingham offspring.PGC-1α dictates endothelial function through regulation of eNOS expressionDecreased neointimal formation in Nox2-deficient mice reveals a direct role for NADPH oxidase in the response to arterial injury.AMP-activated protein kinase: a stress-responsive kinase with implications for cardiovascular disease.Effect of short-term antibiotic treatment on Chlamydia pneumoniae and peripheral endothelial function.
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description
Forscher
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investigador
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onderzoeker
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researcher
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ricercatore
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研究員
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研究者
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name
J F Keaney
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J F Keaney
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J F Keaney
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J F Keaney
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J F Keaney
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J F Keaney
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J F Keaney
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J F Keaney
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J F Keaney
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P106
P31
P496
0000-0002-0866-1837