about
Phosphofructo-1-kinase deficiency leads to a severe cardiac and hematological disorder in addition to skeletal muscle glycogenosisCorrection of diabetic alterations by glucokinase.Prevention of diabetic alterations in transgenic mice overexpressing Myc in the liver.Increased maintenance and persistence of transgenes by excision of expression cassettes from plasmid sequences in vivo.Adipose tissue overexpression of vascular endothelial growth factor protects against diet-induced obesity and insulin resistance.Counteraction of type 1 diabetic alterations by engineering skeletal muscle to produce insulin: insights from transgenic mice.Overexpression of c-myc in the liver prevents obesity and insulin resistance.Prevention of obesity and insulin resistance by glucokinase expression in skeletal muscle of transgenic mice.Long-term overexpression of glucokinase in the liver of transgenic mice leads to insulin resistance.Reversal of type 1 diabetes by engineering a glucose sensor in skeletal muscle.Regulated production of mature insulin by non-beta-cells.Expression of Glucokinase in Skeletal Muscle: A New Approach to Counteract Diabetic HyperglycemiaEvidence from transgenic mice that glucokinase is rate limiting for glucose utilization in the liverEvidence from transgenic mice that myc regulates hepatic glycolysis
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0000-0002-2574-3578