about
PI3K p110δ is expressed by gp38(-)CD31(+) and gp38(+)CD31(+) spleen stromal cells and regulates their CCL19, CCL21, and LTβR mRNA levelsPhosphatidylinositol 3-kinase regulates the CD4/CD8 T cell differentiation ratioPI3K p110γ deletion attenuates murine atherosclerosis by reducing macrophage proliferation but not polarization or apoptosis in lesions.NKG2D ligand overexpression in lupus nephritis correlates with increased NK cell activity and differentiation in kidneys but not in the periphery.Inhibition of natural killer cell activation signals by killer cell immunoglobulin-like receptors (CD158).p38γ and p38δ kinases regulate the Toll-like receptor 4 (TLR4)-induced cytokine production by controlling ERK1/2 protein kinase pathway activationPhosphoinositide 3-kinase gamma participates in T cell receptor-induced T cell activation.Vav1 phosphorylation is induced by beta2 integrin engagement on natural killer cells upstream of actin cytoskeleton and lipid raft reorganization.Cytolytic granule polarization and degranulation controlled by different receptors in resting NK cells.Recent findings on the role of natural killer cells in the pathogenesis of systemic lupus erythematosus.Time-course assessment of the aggregation and metabolization of magnetic nanoparticles.Polyethylenimine-coated SPION exhibits potential intrinsic anti-metastatic properties inhibiting migration and invasion of pancreatic tumor cells.Short-chain PEG molecules strongly bound to magnetic nanoparticle for MRI long circulating agents.Ral GTPases regulate cell-mediated cytotoxicity in NK cells.LFA-1 contributes an early signal for NK cell cytotoxicity.Spontaneous clustering and tyrosine phosphorylation of NK cell inhibitory receptor induced by ligand binding.Coexpression of CD58 or CD48 with intercellular adhesion molecule 1 on target cells enhances adhesion of resting NK cells.Molecular mechanisms of pre-T cell receptor-induced survival.Primary gamma delta cell clones can be defined phenotypically and functionally as Th1/Th2 cells and illustrate the association of CD4 with Th2 differentiation.Statins induce regulatory T cell recruitment via a CCL1 dependent pathway.Polyethylenimine-coated SPIONs trigger macrophage activation through TLR-4 signaling and ROS production and modulate podosome dynamics.T cell receptor diversity in alloreactive responses against HLA-B27 (B*2705) is limited by multiple-level restrictions in both alpha and beta chains.p38γ and p38δ Are Involved in T Lymphocyte Development.Inhibition of PI3Kδ reduces kidney infiltration by macrophages and ameliorates systemic lupus in the mouse.Enhanced phosphoinositide 3-kinase δ activity is a frequent event in systemic lupus erythematosus that confers resistance to activation-induced T cell death.PI3K p85 β regulatory subunit deficiency does not affect NK cell differentiation and increases NKG2D-mediated activation.PTEN Regulation, a Novel Function for the p85 Subunit of Phosphoinositide 3-KinaseMicrothrombotic Renal Vascular Lesions Are Associated to Increased Renal Inflammatory Infiltration in Murine Lupus NephritisMagnetic properties of nanoparticles as a function of their spatial distribution on liposomes and cellsT cells loaded with magnetic nanoparticles are retained in peripheral lymph nodes by the application of a magnetic fieldT-cell receptor usage in alloreactivity against HLA-B*2703 reveals significant conservation of the antigenic structure of B*2705NF-kappa B activation by the pre-T cell receptor serves as a selective survival signal in T lymphocyte developmentMagnetic Nanoparticles Attached to the NK Cell Surface for Tumor Targeting in Adoptive Transfer Therapies Does Not Affect Cellular Effector FunctionsCell-Promoted Nanoparticle Aggregation Decreases Nanoparticle-Induced Hyperthermia under an Alternating Magnetic Field Independently of Nanoparticle Coating, Core Size, and Subcellular LocalizationFlower-like Mn-Doped Magnetic Nanoparticles Functionalized with αvβ3-Integrin-Ligand to Efficiently Induce Intracellular Heat after Alternating Magnetic Field Exposition, Triggering Glioma Cell DeathPolyethylenimine-coated superparamagnetic iron oxide nanoparticles impair in vitro and in vivo angiogenesisMagnetic targeting of adoptively transferred tumour-specific nanoparticle-loaded CD8+ T cells does not improve their tumour infiltration in a mouse model of cancer but promotes the retention of these cells in tumour-draining lymph nodesThe Intrinsic Biological Identities of Iron Oxide Nanoparticles and Their Coatings: Unexplored Territory for Combinatorial Therapies
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P50
description
researcher
@en
name
D F Barber
@en
D F Barber
@nl
type
label
D F Barber
@en
D F Barber
@nl
prefLabel
D F Barber
@en
D F Barber
@nl
P106
P31
P496
0000-0001-8824-5405