about
Engagement of CD160 receptor by HLA-C is a triggering mechanism used by circulating natural killer (NK) cells to mediate cytotoxicity.The transactivating function 1 of estrogen receptor alpha is dispensable for the vasculoprotective actions of 17beta-estradiolMutation of the palmitoylation site of estrogen receptor α in vivo reveals tissue-specific roles for membrane versus nuclear actions.FGF2 translationally induced by hypoxia is involved in negative and positive feedback loops with HIF-1alpha.Changes in Gene Expression and Estrogen Receptor Cistrome in Mouse Liver Upon Acute E2 TreatmentThe uterine and vascular actions of estetrol delineate a distinctive profile of estrogen receptor α modulation, uncoupling nuclear and membrane activation.Determinants of flow-mediated outward remodeling in female rodents: respective roles of age, estrogens, and timing.From in vivo gene targeting of oestrogen receptors to optimization of their modulation in menopause.Estrogens are needed for the improvement in endothelium-mediated dilation induced by a chronic increase in blood flow in rat mesenteric arteries.The Activation Function-1 of Estrogen Receptor Alpha Prevents Arterial Neointima Development Through a Direct Effect on Smooth Muscle Cells.Protective Hematopoietic Effect of Estrogens in a Mouse Model of Thrombosis: Respective Roles of Nuclear Versus Membrane Estrogen Receptor αTherapeutic Benefits and Adverse Effects of Combined Proangiogenic Gene Therapy in Mouse Critical Leg Ischemia.Combined estrogenic and anti-estrogenic properties of estetrol on breast cancer may provide a safe therapeutic window for the treatment of menopausal symptoms.Understanding the oestrogen action in experimental and clinical atherosclerosis.The AF-1-deficient estrogen receptor ERα46 isoform is frequently expressed in human breast tumorsEstrogen receptors and endothelium.Timing of the vascular actions of estrogens in experimental and human studies: why protective early, and not when delayed?Membrane and Nuclear Estrogen Receptor Alpha Actions: From Tissue Specificity to Medical Implications.Stromal estrogen receptor-α promotes tumor growth by normalizing an increased angiogenesis.The short cytoplasmic tail of HLA-G determines its resistance to HIV-1 Nef-mediated cell surface downregulation.Differential down-modulation of HLA-G and HLA-A2 or -A3 cell surface expression following human cytomegalovirus infection.Down-regulation of HLA-G1 cell surface expression in human cytomegalovirus infected cells.Amino acid composition of alpha1/alpha2 domains and cytoplasmic tail of MHC class I molecules determine their susceptibility to human cytomegalovirus US11-mediated down-regulation.Induction of HLA-G-restricted human cytomegalovirus pp65 (UL83)-specific cytotoxic T lymphocytes in HLA-G transgenic mice.Soluble HLA-G: purification from eukaryotic transfected cells and detection by a specific ELISA.Estradiol accelerates endothelial healing through the retrograde commitment of uninjured endothelium.Endothelial estrogen receptor-alpha plays a crucial role in the atheroprotective action of 17beta-estradiol in low-density lipoprotein receptor-deficient mice.Estrogen receptor alpha expression in both endothelium and hematopoietic cells is required for the accelerative effect of estradiol on reendothelialization.Prevention of skin flap necrosis by estradiol involves reperfusion of a protected vascular network.Role of inflammatory cytokines in the effect of estradiol on atheroma.Nuclear and Membrane Actions of Estrogen Receptor Alpha: Contribution to the Regulation of Energy and Glucose Homeostasis.Estetrol, a Fetal Selective Estrogen Receptor Modulator, Acts on the Vagina of Mice through Nuclear Estrogen Receptor α Activation.Testosterone Prevents Cutaneous Ischemia and Necrosis in Males Through Complementary Estrogenic and Androgenic Actions.Cardiovascular benefits and risks of testosterone replacement: another warning?Role of ERαMISS in the Effect of Estradiol on Cancellous and Cortical Femoral Bone in Growing Female MiceRole of estrogens and age in flow-mediated outward remodeling of rat mesenteric resistance arteries.Key role of estrogens and endothelial estrogen receptor α in blood flow-mediated remodeling of resistance arteries.Absence of imprinting of HLA class Ia genes leads to co-expression of biparental alleles on term human trophoblast cells upon IFN-gamma induction.Lymphatic Vasculature Requires ER (Estrogen Receptor)-α Signaling to Protect From Lymphedema.Site-directed mutagenesis of beta-lactamase TEM-1. Investigating the potential role of specific residues on the activity of Pseudomonas-specific enzymes
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P50
description
researcher
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wetenschapper
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F Lenfant
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F Lenfant
@nl
type
label
F Lenfant
@en
F Lenfant
@nl
prefLabel
F Lenfant
@en
F Lenfant
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P106
P31
P496
0000-0003-0024-7241