about
Oral methylthioadenosine administration attenuates fibrosis and chronic liver disease progression in Mdr2-/- miceRegulation of amphiregulin gene expression by β-catenin signaling in human hepatocellular carcinoma cells: a novel crosstalk between FGF19 and the EGFR systemS-adenosylmethionine regulates MAT1A and MAT2A gene expression in cultured rat hepatocytes: a new role for S-adenosylmethionine in the maintenance of the differentiated status of the liverIdentification of argininosuccinate lyase as a hypoxia-responsive gene in rat hepatocytesThe epidermal growth factor receptor: a link between inflammation and liver cancer.Splicing regulator SLU7 preserves survival of hepatocellular carcinoma cells and other solid tumors via oncogenic miR-17-92 cluster expression.Matrix metalloproteinase 10 contributes to hepatocarcinogenesis in a novel crosstalk with the stromal derived factor 1/C-X-C chemokine receptor 4 axis.Ileal FGF15 contributes to fibrosis-associated hepatocellular carcinoma development.Fibroblast Growth Factor 15/19 in Hepatocarcinogenesis.Epidermal growth factor receptor signaling in hepatocellular carcinoma: inflammatory activation and a new intracellular regulatory mechanism.Bile acids, FGF15/19 and liver regeneration: From mechanisms to clinical applications.The epidermal growth factor receptor ligand amphiregulin is a negative regulator of hepatic acute-phase gene expression.Amphiregulin induces the alternative splicing of p73 into its oncogenic isoform DeltaEx2p73 in human hepatocellular tumors.The epidermal growth factor receptor ligand amphiregulin participates in the development of mouse liver fibrosis.5'-methylthioadenosine modulates the inflammatory response to endotoxin in mice and in rat hepatocytes.Liver-specific methionine adenosyltransferase MAT1A gene expression is associated with a specific pattern of promoter methylation and histone acetylation: implications for MAT1A silencing during transformation.Splicing regulator SLU7 is essential for maintaining liver homeostasis.Impairment of pre-mRNA splicing in liver disease: mechanisms and consequences.Inhibition of liver methionine adenosyltransferase gene expression by 3-methylcolanthrene: protective effect of S-adenosylmethionine.Matrix metalloproteinase-10 expression is induced during hepatic injury and plays a fundamental role in liver tissue repair.Lack of Abcc3 expression impairs bile-acid induced liver growth and delays hepatic regeneration after partial hepatectomy in mice.Wilms' tumor 1 gene expression in hepatocellular carcinoma promotes cell dedifferentiation and resistance to chemotherapy.Engineered fibroblast growth factor 19 protects from acetaminophen-induced liver injury and stimulates aged liver regeneration in mice.Prioritizing Popular Proteins in Liver Cancer: Remodelling One-Carbon Metabolism.Fibroblast growth factor 15/19 (FGF15/19) protects from diet-induced hepatic steatosis: development of an FGF19-based chimeric molecule to promote fatty liver regeneration.Toll-like receptor-4 expression by hepatic progenitor cells and biliary epithelial cells in HCV-related chronic liver disease.Splicing events in the control of genome integrity: role of SLU7 and truncated SRSF3 proteinsDNA methylation and histone acetylation of rat methionine adenosyltransferase 1A and 2A genes is tissue-specificHepatocyte growth factor induces MAT2A expression and histone acetylation in rat hepatocytes: role in liver regenerationTransformed but not normal hepatocytes express UCP2Delayed liver regeneration in mice lacking liver serum response factorS-adenosylmethionine regulates cytoplasmic HuR via AMP-activated kinaseDual Targeting of Histone Methyltransferase G9a and DNA-Methyltransferase 1 for the Treatment of Experimental Hepatocellular CarcinomaAlterations in the expression and activity of pre-mRNA splicing factors in hepatocarcinogenesisDefective HNF4alpha-dependent gene expression as a driver of hepatocellular failure in alcoholic hepatitis
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researcher
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wetenschapper
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M U Latasa
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M U Latasa
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M U Latasa
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M U Latasa
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M U Latasa
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M U Latasa
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P106
P31
P496
0000-0002-0878-1337