about
New mutations and horizontal transfer of rpoB among rifampin-resistant Streptococcus pneumoniae from four Spanish hospitalsViridans group streptococci are donors in horizontal transfer of topoisomerase IV genes to Streptococcus pneumoniaeInduction of prophages by fluoroquinolones in Streptococcus pneumoniae: implications for emergence of resistance in genetically-related clones.Role of global and local topology in the regulation of gene expression in Streptococcus pneumoniae.Fluoroquinolone efflux in Streptococcus suis is mediated by SatAB and not by SmrAThe fluoroquinolone levofloxacin triggers the transcriptional activation of iron transport genes that contribute to cell death in Streptococcus pneumoniae.Bridging chromosomal architecture and pathophysiology of Streptococcus pneumoniae.Reactive Oxygen Species Contribute to the Bactericidal Effects of the Fluoroquinolone Moxifloxacin in Streptococcus pneumoniae.HosA, a member of the SlyA family, regulates motility in enteropathogenic Escherichia coli.An increase in negative supercoiling in bacteria reveals topology-reacting gene clusters and a homeostatic response mediated by the DNA topoisomerase I gene.Absence of tmRNA Has a Protective Effect against Fluoroquinolones in Streptococcus pneumoniaeThe genome of Streptococcus pneumoniae is organized in topology-reacting gene clusters.The Transcriptome of Streptococcus pneumoniae Induced by Local and Global Changes in Supercoiling.Nonoptimal DNA topoisomerases allow maintenance of supercoiling levels and improve fitness of Streptococcus pneumoniae.Clinical Isolates of the Spain14-5 clone of Streptococcus pneumoniae carry a recombinant rpoB gene.Epidemiological and molecular aspects of rifampicin-resistant Staphylococcus aureus isolated from wounds, blood and respiratory samples.Upregulation of the PatAB Transporter Confers Fluoroquinolone Resistance to Streptococcus pseudopneumoniae.The promoter of the operon encoding the F0F1 ATPase of Streptococcus pneumoniae is inducible by pH.The membrane-associated F(0)F(1) ATPase is essential for the viability of Streptococcus pneumoniae.HU of Streptococcus pneumoniae Is Essential for the Preservation of DNA Supercoiling.Magnesium-induced linear self-association of the FtsZ bacterial cell division protein monomer. The primary steps for FtsZ assemblyTranscriptional analysis of the acid tolerance response in Streptococcus pneumoniaeAn Uncharacterized Member of the Gls24 Protein Superfamily Is a Putative Sensor of Essential Amino Acid Availability in Streptococcus pneumoniaeBoldine-Derived Alkaloids Inhibit the Activity of DNA Topoisomerase I and Growth of Mycobacterium tuberculosis
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description
researcher
@en
wetenschapper
@nl
name
María José Ferrándiz
@en
María José Ferrándiz
@nl
type
label
María José Ferrándiz
@en
María José Ferrándiz
@nl
prefLabel
María José Ferrándiz
@en
María José Ferrándiz
@nl
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P31
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0000-0003-1428-9506