about
Mutations causing childhood ataxia with central nervous system hypomyelination reduce eukaryotic initiation factor 2B complex formation and activityCandida albicans pathogenicity and epithelial immunityThe discovery of potent, selective, and reversible inhibitors of the house dust mite peptidase allergen Der p 1: an innovative approach to the treatment of allergic asthmaCandidalysin is a fungal peptide toxin critical for mucosal infectionCell cycle-independent phospho-regulation of Fkh2 during hyphal growth regulates Candida albicans pathogenesis.Adaptive immune responses to Candida albicans infection.Candida albicans-epithelial interactions and pathogenicity mechanisms: scratching the surface.Epithelial discrimination of commensal and pathogenic Candida albicans.Protection against epithelial damage during Candida albicans infection is mediated by PI3K/Akt and mammalian target of rapamycin signalingeIF2β is critical for eIF5-mediated GDP-dissociation inhibitor activity and translational control.IL-17 Receptor Signaling in Oral Epithelial Cells Is Critical for Protection against Oropharyngeal Candidiasis.Genome-wide prediction of stop codon readthrough during translation in the yeast Saccharomyces cerevisiae.3-Mercaptopropionic acid-mediated synthesis of peptide and protein thioesters.Critical contacts between the eukaryotic initiation factor 2B (eIF2B) catalytic domain and both eIF2beta and -2gamma mediate guanine nucleotide exchange.Candidalysin drives epithelial signaling, neutrophil recruitment, and immunopathology at the vaginal mucosa.Processing of Candida albicans Ece1p Is Critical for Candidalysin Maturation and Fungal Virulence.Oral epithelial cells orchestrate innate type 17 responses to Candida albicans through the virulence factor candidalysin.Correction to "Production of water-soluble few-layer graphene mesosheets by dry milling with hydrophobic drug".Production of water-soluble few-layer graphene mesosheets by dry milling with hydrophobic drug.Human matrix metalloproteinase-9: activation by limited trypsin treatment and generation of monoclonal antibodies specific for the activated form.Candida-Epithelial Interactions.Candida innate immunity at the mucosa.Candida albicans-Induced Epithelial Damage Mediates Translocation through Intestinal Barriers.Special Issue: Mucosal Fungal Infections.IL-36 and IL-1/IL-17 Drive Immunity to Oral Candidiasis via Parallel MechanismsCandidalysin activates innate epithelial immune responses via epidermal growth factor receptorCandida albicans Interactions with Mucosal Surfaces during Health and DiseaseCandidalysin Is a Potent Trigger of Alarmin and Antimicrobial Peptide Release in Epithelial Cells
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description
researcher
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wetenschapper
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name
Jonathan P Richardson
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Jonathan P Richardson
@nl
type
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Jonathan P Richardson
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Jonathan P Richardson
@nl
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Jonathan P Richardson
@en
Jonathan P Richardson
@nl
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0000-0001-9638-2725