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ANKRD26 and its interacting partners TRIO, GPS2, HMMR and DIPA regulate adipogenesis in 3T3-L1 cellsPartial inactivation of Ankrd26 causes diabetes with enhanced insulin responsiveness of adipose tissue in micePED/PEA-15 inhibits hydrogen peroxide-induced apoptosis in Ins-1E pancreatic beta-cells via PLD-1The GLP-1 receptor agonists exenatide and liraglutide activate Glucose transport by an AMPK-dependent mechanism.Adipose microenvironment promotes triple negative breast cancer cell invasiveness and dissemination by producing CCL5.Methylglyoxal-Glyoxalase 1 Balance: The Root of Vascular Damage.Specific CpG hyper-methylation leads to Ankrd26 gene down-regulation in white adipose tissue of a mouse model of diet-induced obesity.Personalized medicine and type 2 diabetes: lesson from epigenetics.Understanding type 2 diabetes: from genetics to epigenetics.Hoxa5 undergoes dynamic DNA methylation and transcriptional repression in the adipose tissue of mice exposed to high-fat diet.Circulating miRNAs as intercellular messengers, potential biomarkers and therapeutic targets for Type 2 diabetes.Pathologic endoplasmic reticulum stress induced by glucotoxic insults inhibits adipocyte differentiation and induces an inflammatory phenotype.Epigenetics: spotlight on type 2 diabetes and obesity.GRP78 mediates cell growth and invasiveness in endometrial cancer.Targetting PED/PEA-15 for diabetes treatment.Increased levels of the Akt-specific phosphatase PH domain leucine-rich repeat protein phosphatase (PHLPP)-1 in obese participants are associated with insulin resistance.Glucosamine-induced endoplasmic reticulum stress affects GLUT4 expression via activating transcription factor 6 in rat and human skeletal muscle cells.ER stress is associated with dedifferentiation and an epithelial-to-mesenchymal transition-like phenotype in PC Cl3 thyroid cells.Raised expression of the antiapoptotic protein ped/pea-15 increases susceptibility to chemically induced skin tumor development.Platelet-Rich Plasma Increases Growth and Motility of Adipose Tissue-Derived Mesenchymal Stem Cells and Controls Adipocyte Secretory Function.Epigenetic modifications of the Zfp/ZNF423 gene control murine adipogenic commitment and are dysregulated in human hypertrophic obesity.Hepatocyte nuclear factor (HNF)-4alpha-driven epigenetic silencing of the human PED gene.Retraction: ER stress is associated with dedifferentiation and an epithelial-to-mesenchymal transition-like phenotype in PC Cl3 thyroid cells.The role of miR-190a in methylglyoxal-induced insulin resistance in endothelial cells.The PEA15 gene is overexpressed and related to insulin resistance in healthy first-degree relatives of patients with type 2 diabetes.Prep1 deficiency improves metabolic response in white adipose tissue.The Destiny of Glucose from a MicroRNA Perspective.Citrus aurantium L. dry extracts promote C/ebpβ expression and improve adipocyte differentiation in 3T3-L1 cells.Methylglyoxal impairs endothelial insulin sensitivity both in vitro and in vivo.PREP1 deficiency downregulates hepatic lipogenesis and attenuates steatohepatitis in mice.High-fat diet unveils an enhancer element at the Ped/Pea-15 gene responsible for epigenetic memory in skeletal muscleNutritional Factors, DNA Methylation, and Risk of Type 2 Diabetes and Obesity: Perspectives and Challenges
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description
researcher ORCID ID=0000-0003-2742-5634
@en
wetenschapper
@nl
name
G A Raciti
@en
G A Raciti
@nl
type
label
G A Raciti
@en
G A Raciti
@nl
prefLabel
G A Raciti
@en
G A Raciti
@nl
P106
P31
P496
0000-0003-2742-5634