about
Stem cell research in Latin America: update, challenges and opportunities in a priority research areaCell reprogramming and neuronal differentiation applied to neurodegenerative diseases: Focus on Parkinson's diseaseModel based analysis of real-time PCR data from DNA binding dye protocols.Reversible demyelination, blood-brain barrier breakdown, and pronounced neutrophil recruitment induced by chronic IL-1 expression in the brain.Interleukin-1β and tumor necrosis factor-α: reliable targets for protective therapies in Parkinson's Disease?Cell therapy for Parkinson's disease: Functional role of the host immune response on survival and differentiation of dopaminergic neuroblasts.The degenerating substantia nigra as a susceptible region for gene transfer-mediated inflammation.Secreted protein acidic and rich in cysteine produced by human melanoma cells modulates polymorphonuclear leukocyte recruitment and antitumor cytotoxic capacity.Glial Cell-Elicited Activation of Brain Microvasculature in Response to Brucella abortus Infection Requires ASC Inflammasome-Dependent IL-1β Production.Fibulin-2 is a key mediator of the pro-neurogenic effect of TGF-beta1 on adult neural stem cells.Chronic systemic IL-1β exacerbates central neuroinflammation independently of the blood-brain barrier integrity.Prenatal inflammation impairs adult neurogenesis and memory related behavior through persistent hippocampal TGFβ1 downregulation.Nigral neurodegeneration triggered by striatal AdIL-1 administration can be exacerbated by systemic IL-1 expression.Chronic expression of low levels of tumor necrosis factor-alpha in the substantia nigra elicits progressive neurodegeneration, delayed motor symptoms and microglia/macrophage activation.Study of cytokine induced neuropathology by high resolution proton NMR spectroscopy of rat urine.Early and adult hippocampal TGF-β1 overexpression have opposite effects on behavior.Neuronal differentiation in the adult hippocampus recapitulates embryonic development.Learning modulation by endogenous hippocampal IL-1: blockade of endogenous IL-1 facilitates memory formation.A new focal model resembling features of cortical pathology of the progressive forms of multiple sclerosis: influence of innate immunity.Overexpression of IL-1beta by adenoviral-mediated gene transfer in the rat brain causes a prolonged hepatic chemokine response, axonal injury and the suppression of spontaneous behaviour.Special issue commentary: the changing face of inflammation in the brain.CNS response to a second pro-inflammatory event depends on whether the primary demyelinating lesion is active or resolved.Progressive neurodegeneration and motor disabilities induced by chronic expression of IL-1beta in the substantia nigra.Iron Availability Compromises Not Only Oligodendrocytes But Also Astrocytes and Microglial Cells.Chronic Hippocampal Expression of Notch Intracellular Domain Induces Vascular Thickening, Reduces Glucose Availability, and Exacerbates Spatial Memory Deficits in a Rat Model of Early Alzheimer.Current status of stem cells and regenerative medicine research in Argentina.Differential effects of interleukin-1beta on neurotoxicity, cytokine induction and glial reaction in specific brain regions.Patients Beware: Commercialized Stem Cell Treatments on the WebDifferentiation of mesenchymal stem cells into retinal progenitor cellsEnvironmental enrichment improves cognitive symptoms and pathological features in a focal model of cortical damage of multiple sclerosisCell therapy for Parkinson's disease is coming of age: current challenges and future prospects with a focus on immunomodulation
P50
Q26781759-9BDF03D4-E4CA-4B0F-8890-ECAD9C3EE9BEQ28080770-9859F567-1359-4901-AC64-D849852D722CQ31104969-4CECCBB0-AA5B-48A7-AE97-4D8655369003Q35103371-D5D71BE6-54D4-4F0E-9F52-6C0236E2944BQ38103856-9AE0B6F5-AC6B-443A-A405-38BC1D4CC177Q38560345-A9F8159C-5519-4B44-B6BB-964D331EE537Q39737653-C09B6941-7FC1-44EA-A731-5AC92239BE52Q40409041-E99707DC-5B17-45BC-ADB1-7C2D620C1820Q40752714-11D04B7D-3E90-497C-9ABF-FAA544A2CEB1Q40861055-FE4F2F59-6F30-41A9-A6C0-618483530051Q42174806-51413FFF-78E2-43D8-8F15-5B7A3F5D6733Q42987607-A95E7F48-9684-4362-B677-3D3693D09646Q43115655-055F75A2-4FDE-4D97-8747-9A17C482E25FQ43227792-DB7D42DB-1785-437B-9B5B-98343157CAD1Q44936314-53F04A6C-B596-488C-AF18-995884482A72Q46433353-141E5879-2523-4489-AC75-6A050C769BF9Q46788608-E5D51850-356F-4A0C-A3C1-2E1952058BE1Q47270362-7984E943-D173-4EE5-BB77-F99E15B2E1BCQ47834370-BA4F2011-F90B-4187-9E64-611176C8DA55Q48131375-41465E0A-0E0B-4D17-AA1E-5027A12A407CQ48298066-B52C913B-A180-48C7-918D-486CEE4385EFQ48431023-DBAE4022-1948-4906-9A06-10E0796676D8Q48449950-F8777B8A-7C01-48E5-B652-373744A46068Q52678168-F5689552-E35E-4D60-9BD2-3D5454791BE2Q53396776-C0FA44BF-343C-432D-BC50-A57CD2FDEEA1Q53675945-3047606D-DAC0-4E15-B352-9CC5DC8D46C5Q54647613-561AC2B4-7DF7-4E6B-9C1B-F54C7A76EC07Q56896945-F285AEE4-1059-4727-A90B-A9492881EC50Q86277888-C68FC786-F5D0-4C94-8345-84F3E7C3EFCCQ91026687-665741C3-65B9-4437-AE7E-651F08219CC0Q93138854-DDE6ADC3-96A1-4A19-B6B7-C8780C47E37E
P50
description
investigador
@es
researcher
@en
wetenschapper
@nl
name
Fernando J Pitossi
@en
Fernando J Pitossi
@nl
type
label
Fernando J Pitossi
@en
Fernando J Pitossi
@nl
prefLabel
Fernando J Pitossi
@en
Fernando J Pitossi
@nl
P31
P496
0000-0001-5104-9726