about
Contribution of adipocyte-derived factors to beta-cell dysfunction in diabetes.Phenotypic characterization of GPR120-expressing cells in the interstitial tissue of pancreas.Long-term inhibition of protein tyrosine kinase impairs electrophysiologic activity and a rapid component of exocytosis in pancreatic beta-cells.3T3-L1 adipocytes induce dysfunction of MIN6 insulin-secreting cells via multiple pathways mediated by secretory factors in a co-culture system.Activation of ATP-sensitive potassium channels in rat pancreatic beta-cells by linoleic acid through both intracellular metabolites and membrane receptor signalling pathway.Grifolic acid induces mitochondrial membrane potential loss and cell death of RAW264.7 macrophages.The GPR120 Agonist TUG-891 Inhibits the Motility and Phagocytosis of Mouse Alveolar Macrophages
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Q36354022-68232023-28EB-4029-B62C-4761D84BD97EQ39102357-8EF079BF-F668-425E-9C45-233AFA02A880Q40387211-0DFAFF1A-376A-4B46-892A-8C6B589E5788Q42825454-0AE4BEEB-696E-4ADB-A45A-58E9B05C0926Q46542633-9E505AE0-12C8-4068-BFE9-8E83921887AAQ51771839-5253F38B-C834-45C5-9A14-E1F7D67DA7D4Q90151962-BAF9D263-ABB9-45C0-9168-3F388B82444F
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description
investigador
@es
researcher
@en
wetenschapper
@nl
name
Yu-Feng Zhao
@en
Yu-Feng Zhao
@nl
type
label
Yu-Feng Zhao
@en
Yu-Feng Zhao
@nl
prefLabel
Yu-Feng Zhao
@en
Yu-Feng Zhao
@nl
P31
P496
0000-0002-5296-3568