about
Determinants outside the DevR C-terminal domain are essential for cooperativity and robust activation of dormancy genes in Mycobacterium tuberculosis.The Mycobacterium tuberculosis Rv2745c plays an important role in responding to redox stress.Role of TNF in the altered interaction of dormant Mycobacterium tuberculosis with host macrophages.The DosR Regulon Modulates Adaptive Immunity and Is Essential for Mycobacterium tuberculosis Persistence.In-Vivo Gene Signatures of Mycobacterium tuberculosis in C3HeB/FeJ Mice.DosS Is required for the complete virulence of mycobacterium tuberculosis in mice with classical granulomatous lesionsMucosal vaccination with attenuated Mycobacterium tuberculosis induces strong central memory responses and protects against tuberculosisHypoxia Sensing and Persistence Genes Are Expressed during the Intragranulomatous Survival of Mycobacterium tuberculosis.The residue threonine 82 of DevR (DosR) is essential for DevR activation and function in Mycobacterium tuberculosis despite its atypical location.Essentiality of DevR/DosR interaction with SigA for the dormancy survival program in Mycobacterium tuberculosis.Sequencing-relative to hybridization-based transcriptomics approaches better define Mycobacterium tuberculosis stress-response regulons.LAG-3 potentiates the survival of Mycobacterium tuberculosis in host phagocytes by modulating mitochondrial signaling in an in-vitro granuloma model.Characterization of an rpoN mutant of Mesorhizobium ciceri.In vivo inhibition of tryptophan catabolism reorganizes the tuberculoma and augments immune-mediated control of Mycobacterium tuberculosis.
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P50
description
investigador
@es
researcher
@en
wetenschapper
@nl
name
Uma S. Gautam
@en
Uma S. Gautam
@nl
type
label
Uma S. Gautam
@en
Uma S. Gautam
@nl
altLabel
Gautam US
@en
prefLabel
Uma S. Gautam
@en
Uma S. Gautam
@nl
P108
P31
P496
0000-0003-0326-6325