about
Modulating mitochondrial quality in disease transmission: towards enabling mitochondrial DNA disease carriers to have healthy childrenMitophagy plays a central role in mitochondrial ageingIs Placental Mitochondrial Function a Regulator that Matches Fetal and Placental Growth to Maternal Nutrient Intake in the Mouse?Clinical, biochemical, cellular and molecular characterization of mitochondrial DNA depletion syndrome due to novel mutations in the MPV17 geneMitochondrial content is central to nuclear gene expression: Profound implications for human health.The m.13051G>A mitochondrial DNA mutation results in variable neurology and activated mitophagy.Dysregulated mitophagy and mitochondrial organization in optic atrophy due to OPA1 mutations.OPA1 (dys)functions.A novel quantitative assay of mitophagy: Combining high content fluorescence microscopy and mitochondrial DNA load to quantify mitophagy and identify novel pharmacological tools against pathogenic heteroplasmic mtDNA.Correction: Is Placental Mitochondrial Function a Regulator that Matches Fetal and Placental Growth to Maternal Nutrient Intake in the Mouse?Processing of the dynamin Msp1p in S. pombe reveals an evolutionary switch between its orthologs Mgm1p in S. cerevisiae and OPA1 in mammals.Staphylococcal Adhesion and Host Cell Invasion: Fibronectin-Binding and Other Mechanisms.Acute nutritional stress during pregnancy affects placental efficiency, fetal growth and adult glucose homeostasis.Understanding the Virulence of Staphylococcus pseudintermedius: A Major Role of Pore-Forming Toxins.Validating the RedMIT/GFP-LC3 Mouse Model by Studying Mitophagy in Autosomal Dominant Optic Atrophy Due to the OPA1Q285STOP MutationStaphylococcus aureus induces DNA damage in host cellTransmembrane segments of the dynamin Msp1p uncouple its functions in the control of mitochondrial morphology and genome maintenanceEvaluation of the ability of linezolid and tedizolid to eradicate intraosteoblastic and biofilm-embedded Staphylococcus aureus in the bone and joint infection settingUnbiased yeast screens identify cellular pathways affected in Niemann-Pick disease type C
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Q26741333-020ABE1E-4E20-4876-94DF-E45DB64FFA60Q26746323-B498E1D1-02DF-461E-BBB4-A2AA1B55F9F7Q27303878-A59FADA8-6433-46C9-BC0F-EDDB07BEA4E5Q34347182-40A2E39C-DB71-44ED-9707-2D56648F5F36Q36762165-BF6D67C7-E823-412B-B3FA-1981881DCC60Q36918855-1984BFEF-9F17-490F-8DE4-84ADA3814F2EQ37577747-2E32EF7D-80DB-42C2-ABAC-9DD5F74BAEE4Q37665952-717CE05D-ACC9-48FC-89F9-2EBCE11120BFQ41917170-D2BB48F7-4A73-462E-B669-B4C779D68831Q42324164-60B43DCE-5A6A-44B5-A087-7FD33DAC3688Q42973575-973A440F-E521-47E3-B6F7-50B7E1B06132Q47139902-B5AE008B-BA5E-4FB8-8352-088880DC8B07Q47171645-077F1641-DC1A-4B87-BF51-608AE004CC7DQ55645154-8F10B850-3044-4AE3-A9E1-7D9821001154Q57072518-4C252A77-413B-47C0-9B35-1EE2DF5BBD29Q64277770-00E97ABF-9BDB-4873-B68F-265020E43C36Q84147912-BCA9C393-7192-4B3D-A744-8383A5193FF8Q90280488-1F3D06A3-F807-4BE1-AB86-247DDFD273FBQ96125198-10AE5405-29E7-43D7-9AEF-A5A7B377E140
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description
investigador
@es
researcher
@en
wetenschapper
@nl
name
Alan Diot
@en
Alan Diot
@nl
type
label
Alan Diot
@en
Alan Diot
@nl
prefLabel
Alan Diot
@en
Alan Diot
@nl
P31
P496
0000-0001-5079-8274