about
Dendritic cell regulation of NK-cell responses involves lymphotoxin-α, IL-12, and TGF-β.Opposing consequences of signaling through EGF family members: Escape from CTLs could be a bait for NK cellsIL-15 activates mTOR and primes stress-activated gene expression leading to prolonged antitumor capacity of NK cells.Tumor-dependent increase of serum amino acid levels in breast cancer patients has diagnostic potential and correlates with molecular tumor subtypes.Myeloid suppressors decrease melanoma survival by abating tumor-fighting T cells.Tumour-induced immune suppression: role of inflammatory mediators released by myelomonocytic cells.Regulation of myeloid cells by activated T cells determines the efficacy of PD-1 blockade.Ipilimumab treatment results in an early decrease in the frequency of circulating granulocytic myeloid-derived suppressor cells as well as their Arginase1 production.Myeloid-derived suppressor cells impair the quality of dendritic cell vaccines.Tumor-dependent down-regulation of the ζ-chain in T-cells is detectable in early breast cancer and correlates with immune cell function.Targeting Suppressive Myeloid Cells Potentiates Checkpoint Inhibitors to Control Spontaneous Neuroblastoma.Inhibition of tumor-derived prostaglandin-e2 blocks the induction of myeloid-derived suppressor cells and recovers natural killer cell activityHuman ovarian cancer intrinsic mechanisms regulate lymphocyte activation in response to immune checkpoint blockadeWhat industry can teach academiaThe Next Generation of Pattern Recognition Receptor Agonists: Improving Response Rates in Cancer ImmunotherapyCorrection to: Human ovarian cancer intrinsic mechanisms regulate lymphocyte activation in response to immune checkpoint blockade
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description
investigador
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researcher
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Y Mao
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Y Mao
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Y Mao
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P108
P31
P496
0000-0001-6142-6628