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NaCl cotransporter abundance in urinary vesicles is increased by calcineurin inhibitors and predicts thiazide sensitivity.Genetic ablation of aquaporin-2 in the mouse connecting tubules results in defective renal water handling.A Systems Level Analysis of Vasopressin-mediated Signaling Networks in Kidney Distal Convoluted Tubule Cells.Renal aquaporins and water balance disorders.Vasopressin regulation of sodium transport in the distal nephron and collecting duct.Hypotonicity-induced reduction of aquaporin-2 transcription in mpkCCD cells is independent of the tonicity responsive element, vasopressin, and cAMPEffect of the cGMP pathway on AQP2 expression and translocation: potential implications for nephrogenic diabetes insipidus.Amiloride blocks lithium entry through the sodium channel thereby attenuating the resultant nephrogenic diabetes insipidus.Phosphorylation decreases ubiquitylation of the thiazide-sensitive cotransporter NCC and subsequent clathrin-mediated endocytosis.Demeclocycline attenuates hyponatremia by reducing aquaporin-2 expression in the renal inner medulla.Hydrochlorothiazide attenuates lithium-induced nephrogenic diabetes insipidus independently of the sodium-chloride cotransporter.CHIP Regulates Aquaporin-2 Quality Control and Body Water Homeostasis.Lithium induces aerobic glycolysis and glutaminolysis in collecting duct principal cells.Counteracting vasopressin-mediated water reabsorption by ATP, dopamine, and phorbol esters: mechanisms of actionIn mpkCCD cells, long-term regulation of aquaporin-2 by vasopressin occurs independent of protein kinase A and CREB but may involve Epac
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description
investigador
@es
researcher
@en
wetenschapper
@nl
name
Marleen L A Kortenoeven
@en
Marleen L A Kortenoeven
@nl
type
label
Marleen L A Kortenoeven
@en
Marleen L A Kortenoeven
@nl
prefLabel
Marleen L A Kortenoeven
@en
Marleen L A Kortenoeven
@nl
P31
P496
0000-0001-9962-8276