about
Mouse hepatocyte overexpression of NF-κB-inducing kinase (NIK) triggers fatal macrophage-dependent liver injury and fibrosis.Myeloid cell TRAF3 promotes metabolic inflammation, insulin resistance, and hepatic steatosis in obesity.Lipocalin 13 protein protects against hepatic steatosis by both inhibiting lipogenesis and stimulating fatty acid β-oxidation.Carboxyl terminus of HSC70-interacting protein (CHIP) down-regulates NF-κB-inducing kinase (NIK) and suppresses NIK-induced liver injuryHepatic TRAF2 regulates glucose metabolism through enhancing glucagon responses.Herbal constituent sequoyitol improves hyperglycemia and glucose intolerance by targeting hepatocytes, adipocytes, and β-cellsNF-κB–inducing kinase (NIK) promotes hyperglycemia and glucose intolerance in obesity by augmenting glucagon actionAdipose Snail1 Regulates Lipolysis and Lipid Partitioning by Suppressing Adipose Triacylglycerol Lipase Expression.Liver NF-κB-inducing Kinase (NIK) Promotes Liver Steatosis and Glucose Counterregulation in Male Mice with Obesity.Thymic NF-κB-inducing kinase regulates CD4+ T cell-elicited liver injury and fibrosis in mice.The hepatokine Tsukushi gates energy expenditure via brown fat sympathetic innervationLeptin receptor-expressing neuron Sh2b1 supports sympathetic nervous system and protects against obesity and metabolic diseaseBrown fat activation mitigates alcohol-induced liver steatosis and injury in miceMedullary thymic epithelial NF-kB-inducing kinase (NIK)/IKKα pathway shapes autoimmunity and liver and lung homeostasis in mice
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description
researcher (ORCID 0000-0002-1559-3633)
@en
name
Hong Shen
@en
type
label
Hong Shen
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prefLabel
Hong Shen
@en
P31
P496
0000-0002-1559-3633