about
Overexpression of Dyrk1A is implicated in several cognitive, electrophysiological and neuromorphological alterations found in a mouse model of Down syndromePre- and post-natal melatonin administration partially regulates brain oxidative stress but does not improve cognitive or histological alterations in the Ts65Dn mouse model of Down syndrome.Inflammatory dysregulation of monocytes in pediatric patients with obsessive-compulsive disorder.Microarray gene-expression study in fibroblast and lymphoblastoid cell lines from antipsychotic-naïve first-episode schizophrenia patients.Decreasing the Expression of GABAA α5 Subunit-Containing Receptors Partially Improves Cognitive, Electrophysiological, and Morphological Hippocampal Defects in the Ts65Dn Model of Down Syndrome.Cerebellar alterations in a model of Down syndrome: The role of the Dyrk1A gene.Normalizing the gene dosage of Dyrk1A in a mouse model of Down syndrome rescues several Alzheimer's disease phenotypes.Chronic Melatonin Administration Reduced Oxidative Damage and Cellular Senescence in the Hippocampus of a Mouse Model of Down Syndrome.Anti-IL17 treatment ameliorates Down syndrome phenotypes in mice
P50
Q34133976-FAA3E0CA-544C-4358-85E3-B002E10E0AC3Q47306676-F6A0A6F7-1F2D-4037-8CC0-4522E44B9E1BQ47560028-4AD6FD8A-ECFE-46C6-9827-5648E0558CF3Q47642710-B801122D-9C69-4D8A-A77D-EC36820F752DQ47666263-5DB4E1A7-3186-4DB1-9DE8-62AA4E1BE904Q47925844-EB0A9565-5BAE-4C01-B135-D44C7340B34CQ48225546-6B31DECC-74A9-458A-8E10-D19A28F24CB9Q48444907-646FBA2D-B9D2-49E3-AF4F-81B01E411AF4Q88664424-17DCE286-F8A5-458E-8FCF-A981DF609B62
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description
investigador
@es
researcher
@en
wetenschapper
@nl
name
Susana García-Cerro
@en
Susana García-Cerro
@nl
type
label
Susana García-Cerro
@en
Susana García-Cerro
@nl
prefLabel
Susana García-Cerro
@en
Susana García-Cerro
@nl
P31
P496
0000-0002-6091-2824