about
CYTOKININ OXIDASE/DEHYDROGENASE3 Maintains Cytokinin Homeostasis during Root and Nodule Development in Lotus japonicus.A legume genetic framework controls infection of nodules by symbiotic and endophytic bacteria.Differential regulation of the Epr3 receptor coordinates membrane-restricted rhizobial colonization of root nodule primordia.Structures of Exopolysaccharides Involved in Receptor-mediated Perception of Mesorhizobium loti by Lotus japonicus.The Ethylene Responsive Factor Required for Nodulation 1 (ERN1) Transcription Factor Is Required for Infection-Thread Formation in Lotus japonicus.Receptor-mediated exopolysaccharide perception controls bacterial infection.Conditional requirement for exopolysaccharide in the Mesorhizobium-Lotus symbiosis.The Brassicaceae family displays divergent, shoot-skewed NLR resistance gene expression.Regulation of Nod factor biosynthesis by alternative NodD proteins at distinct stages of symbiosis provides additional compatibility scrutiny.Dynamics of Lotus japonicus ethylene production in response to compatible Nod factor.Legume LysM receptors mediate symbiotic and pathogenic signalling.Epidermal auxin biosynthesis facilitates rhizobial infection in Lotus japonicus.Epidermal LysM receptor ensures robust symbiotic signalling in Lotus japonicus.A genetic screen for plant mutants with altered nodulation phenotypes in response to rhizobial glycan mutantsA plant chitinase controls cortical infection thread progression and nitrogen-fixing symbiosisDistinct Transcriptomic Responses to a Spectrum of Bacteria Ranging From Symbiotic to PathogenicSinorhizobium fredii HH103 nolR and nodD2 mutants gain capacity for infection thread invasion of Lotus japonicus Gifu and Lotus burttii
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description
investigador
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researcher
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name
Simon Kelly
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type
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Simon Kelly
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Simon Kelly
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0000-0003-4243-300X