about
Mitochondrial alarmins released by degenerating motor axon terminals activate perisynaptic Schwann cellsSnake and Spider Toxins Induce a Rapid Recovery of Function of Botulinum Neurotoxin Paralysed Neuromuscular JunctionBotulinum neurotoxin C mutants reveal different effects of syntaxin or SNAP-25 proteolysis on neuromuscular transmission.CXCL12α/SDF-1 from perisynaptic Schwann cells promotes regeneration of injured motor axon terminals.An animal model of Miller Fisher syndrome: Mitochondrial hydrogen peroxide is produced by the autoimmune attack of nerve terminals and activates Schwann cells.Calpains participate in nerve terminal degeneration induced by spider and snake presynaptic neurotoxins.
P50
description
investigador
@es
researcher
@en
name
Elisa Duregotti
@en
type
label
Elisa Duregotti
@en
prefLabel
Elisa Duregotti
@en
P31
P496
0000-0003-1409-4063