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Key Events Participating in the Pathogenesis of Alcoholic Liver Disease.Rationale for the use of statins in liver disease.Signalling via the osteopontin and high mobility group box-1 axis drives the fibrogenic response to liver injury."Tipping" extracellular matrix remodeling towards regression of liver fibrosis: novel concepts.Cartilage oligomeric matrix protein participates in the pathogenesis of liver fibrosis.Osteopontin deletion drives hematopoietic stem cell mobilization to the liver and increases hepatic iron contributing to alcoholic liver disease.Circulating microbiome in blood of different circulatory compartmentsHigh Mobility Group Box-1 Drives Fibrosis Progression Signaling via the Receptor for Advanced Glycation End Products in MiceRho-kinase inhibitor coupled to peptide-modified albumin carrier reduces portal pressure and increases renal perfusion in cirrhotic ratsSelective LOXL2 inhibition: potent antifibrotic effects in ongoing fibrosis and fibrosis regressionTGR(mREN2)27 rats develop non-alcoholic fatty liver disease-associated portal hypertension responsive to modulations of Janus-kinase 2 and Mas receptor
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description
investigador
@es
researcher
@en
wetenschapper
@nl
name
Fernando Magdaleno
@en
Fernando Magdaleno
@nl
type
label
Fernando Magdaleno
@en
Fernando Magdaleno
@nl
prefLabel
Fernando Magdaleno
@en
Fernando Magdaleno
@nl
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P496
0000-0002-4121-6743