about
Molecular basis for the genome engagement by Sox proteinsCervical cancer stem cells manifest radioresistance: Association with upregulated AP-1 activity.Deregulation of microRNAs Let-7a and miR-21 mediate aberrant STAT3 signaling during human papillomavirus-induced cervical carcinogenesis: role of E6 oncoprotein.Cervical cancer screening and treatment of cervical intraepithelial neoplasia in female sex workers using "screen and treat" approach.Alterations in microRNAs miR-21 and let-7a correlate with aberrant STAT3 signaling and downstream effects during cervical carcinogenesis.Structure and decoy-mediated inhibition of the SOX18/Prox1-DNA interactionQuantitative profiling of selective Sox/POU pairing on hundreds of sequences in parallel by Coop-seq.The Crystal Structure of a Maxi/Mini-Ferritin Chimera Reveals Guiding Principles for the Assembly of Protein Cages.Cervical Cancer Stem Cells Selectively Overexpress HPV Oncoprotein E6 that Controls Stemness and Self-Renewal through Upregulation of HES1.Coop-Seq Analysis Demonstrates that Sox2 Evokes Latent Specificities in the DNA Recognition by Pax6.Mutations involving the SRY-related gene SOX8 are associated with a spectrum of human reproductive anomalies.DNA-mediated dimerization on a compact sequence signature controls enhancer engagement and regulation by FOXA1.Directed Evolution of Reprogramming Factors by Cell Selection and SequencingPluripotency reprogramming by competent and incompetent POU factors uncovers temporal dependency for Oct4 and Sox2
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P50
description
investigador
@es
researcher
@en
name
Yogesh Srivastava
@en
type
label
Yogesh Srivastava
@en
prefLabel
Yogesh Srivastava
@en
P31
P496
0000-0002-2370-3767