about
Synthetic lethality interaction between Aurora kinases and CHEK1 inhibitors in ovarian cancer.Targeting basal-like breast tumors with bromodomain and extraterminal domain (BET) and polo-like kinase inhibitors.In silico analyses identify gene-sets, associated with clinical outcome in ovarian cancer: role of mitotic kinases.Neutrophils in cancer: prognostic role and therapeutic strategies.Mitotic read-out genes confer poor outcome in luminal A breast cancer tumors.DNA-damage related genes and clinical outcome in hormone receptor positive breast cancerBET inhibitors as novel therapeutic agents in breast cancer.Evaluation of transcriptionally regulated genes identifies NCOR1 in hormone receptor negative breast tumors and lung adenocarcinomas as a potential tumor suppressor geneMapping Bromodomains in breast cancer and association with clinical outcomeGenetic mutational status of genes regulating epigenetics: Role of the histone methyltransferase KMT2D in triple negative breast tumorsExpression of MHC class I, HLA-A and HLA-B identifies immune-activated breast tumors with favorable outcomeTrastuzumab-Targeted Biodegradable Nanoparticles for Enhanced Delivery of Dasatinib in HER2+ Metastasic Breast CancerScreening and Preliminary Biochemical and Biological Studies of [RuCl(p-cymene)(N,N-bis(diphenylphosphino)-isopropylamine)][BF4] in Breast Cancer ModelsPoly(Cyclohexene Phthalate) Nanoparticles for Controlled Dasatinib Delivery in Breast Cancer TherapyActivity of BET-proteolysis targeting chimeric (PROTAC) compounds in triple negative breast cancer
P50
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P50
description
investigador
@es
researcher
@en
wetenschapper
@nl
name
Cristina Nieto-Jiménez
@en
Cristina Nieto-Jiménez
@nl
type
label
Cristina Nieto-Jiménez
@en
Cristina Nieto-Jiménez
@nl
prefLabel
Cristina Nieto-Jiménez
@en
Cristina Nieto-Jiménez
@nl
P31
P496
0000-0002-3463-1439