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Tau acts as a mediator for Alzheimer's disease-related synaptic deficits.Tau phosphorylation and tau mislocalization mediate soluble Aβ oligomer-induced AMPA glutamate receptor signaling deficits.A53T Mutant Alpha-Synuclein Induces Tau-Dependent Postsynaptic Impairment Independently of Neurodegenerative Changes.Tau is required for progressive synaptic and memory deficits in a transgenic mouse model of α-synucleinopathyPhosphorylation in two discrete tau domains regulates a stepwise process leading to postsynaptic dysfunctionThe Accumulation of Tau in Postsynaptic Structures: A Common Feature in Multiple Neurodegenerative Diseases?
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description
investigador
@es
researcher
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wetenschapper
@nl
name
Peter J Teravskis
@en
Peter J Teravskis
@nl
type
label
Peter J Teravskis
@en
Peter J Teravskis
@nl
prefLabel
Peter J Teravskis
@en
Peter J Teravskis
@nl
P31
P496
0000-0002-3204-8389