about
Sporadic granular cell tumours lack recurrent mutations in PTPN11, PTEN and other cancer-related genes.TRPV4 and KRAS and FGFR1 gain-of-function mutations drive giant cell lesions of the jawLoss of heterozygosity of MIR15A/MIR16-1 , negative regulators of the antiapoptotic gene BCL2 , is not common in odontogenic keratocystsKRAS mutations drive adenomatoid odontogenic tumor and are independent of clinicopathological featuresMaking sense of giant cell lesions of the jaws (GCLJ): lessons learned from next-generation sequencing
P50
description
researcher
@en
name
Marina Gonçalves Diniz
@en
type
label
Marina Gonçalves Diniz
@en
prefLabel
Marina Gonçalves Diniz
@en
P31
P496
0000-0002-4212-1172