about
Regulatory mechanisms that prevent re-initiation of DNA replication can be locally modulated at origins by nearby sequence elements.Non-homologous DNA increases gene disruption efficiency by altering DNA repair outcomes.Enhancing homology-directed genome editing by catalytically active and inactive CRISPR-Cas9 using asymmetric donor DNA.CRISPR–Cas9 genome editing in human cells occurs via the Fanconi anemia pathwayUnbiased detection of CRISPR off-targets in vivo using DISCOVER-SeqAdvances in genome editing through control of DNA repair pathwaysTimed inhibition of CDC7 increases CRISPR-Cas9 mediated templated repairSuppression of unwanted CRISPR-Cas9 editing by co-administration of catalytically inactivating truncated guide RNAs
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Q35191354-1220C2BF-EEBB-4462-80DA-08453E047184Q38751553-7BAF3113-877F-4E93-B467-E430DC338D06Q38801430-88A53B0C-F6D1-4876-BBE7-166F79D55B9EQ58054240-C9644BE5-3400-48CA-BD0D-4A44E8C336E5Q64374718-39A82FED-60C7-4EA1-B667-57A3257999EDQ91620907-E9179AA6-4B20-4C7C-9486-B477E049436CQ94474051-BFDEDE97-931F-4687-814D-2981080A739BQ96126089-06C3212D-825C-446C-8367-671C986B43EF
P50
description
researcher
@en
wetenschapper
@nl
name
Christopher D Richardson
@en
Christopher D Richardson
@nl
type
label
Christopher D Richardson
@en
Christopher D Richardson
@nl
prefLabel
Christopher D Richardson
@en
Christopher D Richardson
@nl
P31
P496
0000-0003-3122-4851