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Postnatal Zika virus infection is associated with persistent abnormalities in brain structure, function, and behavior in infant macaques.Short-term pIFN-α2a treatment does not significantly reduce the viral reservoir of SIV-infected, ART-treated rhesus macaques.Antibody-Mediated CD4 Depletion Induces Homeostatic CD4 T Cell Proliferation without Detectable Virus Reactivation in Antiretroviral Therapy-Treated Simian Immunodeficiency Virus-Infected MacaquesPharmacological Modulation of the Wnt/β-Catenin Pathway Inhibits Proliferation and Promotes Differentiation of Long-Lived Memory CD4+ T Cells in Antiretroviral Therapy-Suppressed Simian Immunodeficiency Virus-Infected MacaquesCorrection for Mavigner et al., "Simian Immunodeficiency Virus Persistence in Cellular and Anatomic Reservoirs in Antiretroviral Therapy-Suppressed Infant Rhesus Macaques"Analytical Treatment Interruption after Short-Term Antiretroviral Therapy in a Postnatally Simian-Human Immunodeficiency Virus-Infected Infant Rhesus Macaque Model
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description
researcher
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name
Maud Mavigner
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type
label
Maud Mavigner
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prefLabel
Maud Mavigner
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P496
0000-0001-9898-317X