about
Epstein-Barr virus fusion with epithelial cells triggered by gB is restricted by a gL glycosylation site.Ephrin receptor A2 is a functional entry receptor for Epstein-Barr virus.EBV latent membrane protein 2A orchestrates p27kip1 degradation via Cks1 to accelerate MYC-driven lymphoma in mice.Natural Selection of Glycoprotein B Mutations That Rescue the Small-Plaque Phenotype of a Fusion-Impaired Herpes Simplex Virus MutantSpleen Tyrosine Kinase Inhibitor TAK-659 Prevents Splenomegaly and Tumor Development in a Murine Model of Epstein-Barr Virus-Associated LymphomaErratum for Riccio et al., "Characterization of Sex Differences in Ocular Herpes Simplex Virus 1 Infection and Herpes Stromal Keratitis Pathogenesis of Wild-Type and Herpesvirus Entry Mediator Knockout Mice"Two Pathways of p27 Degradation Are Required for Murine Lymphoma Driven by Myc and EBV Latent Membrane Protein 2ACharacterization of Sex Differences in Ocular Herpes Simplex Virus 1 Infection and Herpes Stromal Keratitis Pathogenesis of Wild-Type and Herpesvirus Entry Mediator Knockout MiceEphrin Receptor A4 is a New Kaposi's Sarcoma-Associated Herpesvirus Virus Entry ReceptorHerpesvirus Entry Mediator Binding Partners Mediate Immunopathogenesis of Ocular Herpes Simplex Virus 1 InfectionThe Innate Immune Response to Herpes Simplex Virus 1 Infection Is Dampened in the Newborn Brain and Can Be Modulated by Exogenous Interferon Beta To Improve Survival
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description
researcher
@en
name
Richard Longnecker
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type
label
Richard Longnecker
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prefLabel
Richard Longnecker
@en
P31
P496
0000-0001-7175-6217