about
Absence of physiological Ca2+ transients is an initial trigger for mitochondrial dysfunction in skeletal muscle following denervation.Irisin protects mitochondria function during pulmonary ischemia/reperfusion injury.Sustained elevation of MG53 in the bloodstream increases tissue regenerative capacity without compromising metabolic function
P50
description
researcher (ORCID 0000-0002-5684-7177)
@en
name
Jingsong Zhou
@en
type
label
Jingsong Zhou
@en
prefLabel
Jingsong Zhou
@en
P31
P496
0000-0002-5684-7177