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Q33289279-1CA1A68E-E9CD-4E97-AC2A-828A42E537AA
Q33289279-1CA1A68E-E9CD-4E97-AC2A-828A42E537AA
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Statement
http://www.wikidata.org/entity/statement/Q33289279-1CA1A68E-E9CD-4E97-AC2A-828A42E537AA
Probing mechanisms of resistance to the tuberculosis drug isoniazid: Conformational changes caused by inhibition of InhA, the enoyl reductase from Mycobacterium tuberculosis.
P2860
Q33289279-1CA1A68E-E9CD-4E97-AC2A-828A42E537AA
BestRank
Statement
http://www.wikidata.org/entity/statement/Q33289279-1CA1A68E-E9CD-4E97-AC2A-828A42E537AA
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type
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wasDerivedFrom
c921ddb90cd63592d9195a009a3686ccd7533535
P2860
InhA, a target of the antituberculous drug isoniazid, is involved in a mycobacterial fatty acid elongation system, FAS-II