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Q35311053-090B2B97-344D-42F3-8459-4FE3A84CA1D4
Q35311053-090B2B97-344D-42F3-8459-4FE3A84CA1D4
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http://www.wikidata.org/entity/statement/Q35311053-090B2B97-344D-42F3-8459-4FE3A84CA1D4
Proteasomal dysfunction and endoplasmic reticulum stress enhance trafficking of prion protein aggregates through the secretory pathway and increase accumulation of pathologic prion protein
P2860
Q35311053-090B2B97-344D-42F3-8459-4FE3A84CA1D4
BestRank
Statement
http://www.wikidata.org/entity/statement/Q35311053-090B2B97-344D-42F3-8459-4FE3A84CA1D4
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wasDerivedFrom
5758aff3f49f98c731cee8d9e6c2cfa36580a0ca
P2860
Mutant prion proteins are partially retained in the endoplasmic reticulum.