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Q51736555-6FA68381-EBA4-49D4-8298-E2B6B9282075
Q51736555-6FA68381-EBA4-49D4-8298-E2B6B9282075
BestRank
Statement
http://www.wikidata.org/entity/statement/Q51736555-6FA68381-EBA4-49D4-8298-E2B6B9282075
Therapeutic Genome Editing With CRISPR/Cas9 in a Humanized Mouse Model Ameliorates α1-antitrypsin Deficiency Phenotype.
P2860
Q51736555-6FA68381-EBA4-49D4-8298-E2B6B9282075
BestRank
Statement
http://www.wikidata.org/entity/statement/Q51736555-6FA68381-EBA4-49D4-8298-E2B6B9282075
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wasDerivedFrom
cf9cb8c6f25e2949e1b2f17a57dd2a363dc58481
P2860
The endoplasmic reticulum degradation pathway for mutant secretory proteins alpha1-antitrypsin Z and S is distinct from that for an unassembled membrane protein.