Neuronal nitric oxide synthase control mechanisms in the cutaneous vasculature of humans in vivo
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Does skin moisture influence the blood flow response to local heat? A re-evaluation of the Pennes modelLocal thermal control of the human cutaneous circulationEndothelial nitric oxide synthase control mechanisms in the cutaneous vasculature of humans in vivoExercise prevents age-related decline in nitric-oxide-mediated vasodilator function in cutaneous microvessels.The involvement of norepinephrine, neuropeptide Y, and nitric oxide in the cutaneous vasodilator response to local heating in humans.Nonselective NOS inhibition blunts the sweat response to exercise in a warm environment.The effect of microdialysis needle trauma on cutaneous vascular responses in humans.Sustained impairments in cutaneous vasodilation and sweating in grafted skin following long-term recoveryRoles of nitric oxide synthase isoforms in cutaneous vasodilation induced by local warming of the skin and whole body heat stress in humans.Plasma hyperosmolality elevates the internal temperature threshold for active thermoregulatory vasodilation during heat stress in humans.Cutaneous vascular and sudomotor responses in human skin grafts.Transient receptor potential vanilloid type-1 (TRPV-1) channels contribute to cutaneous thermal hyperaemia in humans.Heat acclimation improves cutaneous vascular function and sweating in trained cyclists.Antagonism of soluble guanylyl cyclase attenuates cutaneous vasodilation during whole body heat stress and local warming in humans.Limb-specific differences in the skin vascular responsiveness to adrenergic agonists.Ascorbic acid or L-arginine improves cutaneous microvascular function in chronic kidney disease.Upregulation of inducible nitric oxide synthase contributes to attenuated cutaneous vasodilation in essential hypertensive humans.Local tetrahydrobiopterin administration augments reflex cutaneous vasodilation through nitric oxide-dependent mechanisms in aged human skin.Mechanisms and modifiers of reflex induced cutaneous vasodilation and vasoconstriction in humans.Transient receptor potential vanilloid type 1 channels contribute to reflex cutaneous vasodilation in humans.5-hydroxytryptamine (5-HT) reduces total peripheral resistance during chronic infusion: direct arterial mesenteric relaxation is not involved.KCa channels and epoxyeicosatrienoic acids: major contributors to thermal hyperaemia in human skin.Neuronal nitric oxide synthase and human vascular regulationNitric oxide and receptors for VIP and PACAP in cutaneous active vasodilation during heat stress in humans.Mechanisms underlying the postexercise baroreceptor-mediated suppression of heat loss.Is the effect of mobile phone radiofrequency waves on human skin perfusion non-thermal?No independent, but an interactive, role of calcium-activated potassium channels in human cutaneous active vasodilation.Noradrenaline and neuropeptide Y contribute to initial, but not sustained, vasodilatation in response to local skin warming in humans.Modelling of thermal hyperemia in the skin of type 2 diabetic patients.Endothelial nitric oxide synthase mediates the nitric oxide component of reflex cutaneous vasodilatation during dynamic exercise in humans.Comparison of the noradrenergic sympathetic nerve contribution during local skin heating at forearm and leg sites in humans.Cutaneous vascular and sweating responses to intradermal administration of ATP: a role for nitric oxide synthase and cyclooxygenase?Augmented reflex cutaneous vasodilatation following short-term dietary nitrate supplementation in humans.Microvascular reactivity to thermal stimulation in patients with diabetes mellitus and polyneuropathy.iNOS-dependent sweating and eNOS-dependent cutaneous vasodilation are evident in younger adults, but are diminished in older adults exercising in the heatROS and endothelial nitric oxide synthase (eNOS)-dependent trafficking of angiotensin II type 2 receptor begets neuronal NOS in cardiac myocytes.Sympathetic activation increases NO release from eNOS but neither eNOS nor nNOS play an essential role in exercise hyperemia in the human forearm.TNF signals via neuronal-type nitric oxide synthase and reactive oxygen species to depress specific force of skeletal muscle.Adrenergic control of the human cutaneous circulation.Current concepts of active vasodilation in human skin.
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P2860
Neuronal nitric oxide synthase control mechanisms in the cutaneous vasculature of humans in vivo
description
2008 nî lūn-bûn
@nan
2008 թուականի Փետրուարին հրատարակուած գիտական յօդուած
@hyw
2008 թվականի փետրվարին հրատարակված գիտական հոդված
@hy
2008年の論文
@ja
2008年論文
@yue
2008年論文
@zh-hant
2008年論文
@zh-hk
2008年論文
@zh-mo
2008年論文
@zh-tw
2008年论文
@wuu
name
Neuronal nitric oxide synthase ...... vasculature of humans in vivo
@ast
Neuronal nitric oxide synthase ...... vasculature of humans in vivo
@en
Neuronal nitric oxide synthase ...... vasculature of humans in vivo
@en-gb
Neuronal nitric oxide synthase ...... vasculature of humans in vivo
@nl
type
label
Neuronal nitric oxide synthase ...... vasculature of humans in vivo
@ast
Neuronal nitric oxide synthase ...... vasculature of humans in vivo
@en
Neuronal nitric oxide synthase ...... vasculature of humans in vivo
@en-gb
Neuronal nitric oxide synthase ...... vasculature of humans in vivo
@nl
prefLabel
Neuronal nitric oxide synthase ...... vasculature of humans in vivo
@ast
Neuronal nitric oxide synthase ...... vasculature of humans in vivo
@en
Neuronal nitric oxide synthase ...... vasculature of humans in vivo
@en-gb
Neuronal nitric oxide synthase ...... vasculature of humans in vivo
@nl
P2093
P2860
P3181
P1476
Neuronal nitric oxide synthase ...... vasculature of humans in vivo
@en
P2093
Dean L Kellogg
Joan L Zhao
P2860
P304
P3181
P356
10.1113/JPHYSIOL.2007.144642
P407
P577
2008-02-01T00:00:00Z