CCM1 regulates vascular-lumen organization by inducing endothelial polarity
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"You Shall Not Pass"-tight junctions of the blood brain barrierMolecular control of capillary morphogenesis and maturation by recognition and remodeling of the extracellular matrix: functional roles of endothelial cells and pericytes in health and diseaseCerebral cavernous malformation is a vascular disease associated with activated RhoA signalingMolecular mechanisms controlling vascular lumen formation in three-dimensional extracellular matricesLack of CCM1 induces hypersprouting and impairs response to flowActin remodeling by Nck regulates endothelial lumen formationThe alternative splicing factor Nova2 regulates vascular development and lumen formation.Cdc42 and k-Ras Control Endothelial Tubulogenesis through Apical Membrane and Cytoskeletal Polarization: Novel Stimulatory Roles for GTPase Effectors, the Small GTPases, Rac2 and Rap1b, and Inhibitory Influence of Arhgap31 and Rasa1Single-cell analysis of endothelial morphogenesis in vivo.Structural basis of the junctional anchorage of the cerebral cavernous malformations complexThe structure of Rap1 in complex with RIAM reveals specificity determinants and recruitment mechanismBlood vessel tubulogenesis requires Rasip1 regulation of GTPase signalingWnt activation of immortalized brain endothelial cells as a tool for generating a standardized model of the blood brain barrier in vitroSpatial regulation of VEGF receptor endocytosis in angiogenesisKLF4 is a key determinant in the development and progression of cerebral cavernous malformations.An apical actin-rich domain drives the establishment of cell polarity during cell adhesion.Biomimetic model to reconstitute angiogenic sprouting morphogenesis in vitro.VE-PTP regulates VEGFR2 activity in stalk cells to establish endothelial cell polarity and lumen formationCCM1-ICAP-1 complex controls β1 integrin-dependent endothelial contractility and fibronectin remodelingPresymptomatic activation of the PDGF-CC pathway accelerates onset of ALS neurodegeneration.VE-cadherin interacts with cell polarity protein Pals1 to regulate vascular lumen formation.Endothelial adherens junctions and the actin cytoskeleton: an 'infinity net'?CD47 deficiency in tumor stroma promotes tumor progression by enhancing angiogenesis.Endothelial permeability and VE-cadherin: a wacky comradeship.Enhanced sphingosine-1-phosphate receptor 2 expression underlies female CNS autoimmunity susceptibility.RhoJ is an endothelial cell-restricted Rho GTPase that mediates vascular morphogenesis and is regulated by the transcription factor ERG.Developmental timing of CCM2 loss influences cerebral cavernous malformations in mice.Endothelial deficiency of L1 reduces tumor angiogenesis and promotes vessel normalization.Multiple endothelial cells constitute the tip of developing blood vessels and polarize to promote lumen formation.A distinct mechanism of vascular lumen formation in Xenopus requires EGFL7Endothelial cell hyperproliferation and stratification in uteroplacental blood vessels of the black mastiff bat, Molossus rufusSulindac metabolites decrease cerebrovascular malformations in CCM3-knockout mice.A VE-cadherin-PAR3-α-catenin complex regulates the Golgi localization and activity of cytosolic phospholipase A(2)α in endothelial cells.Rap1 GTPase is required for mouse lens epithelial maintenance and morphogenesis.PDCD10 (CCM3) regulates brain endothelial barrier integrity in cerebral cavernous malformation type 3: role of CCM3-ERK1/2-cortactin cross-talk.Mass-Selected Site-Specific Core-Fucosylation of Serum Proteins in Hepatocellular Carcinoma.Formation of a PKCζ/β-catenin complex in endothelial cells promotes angiopoietin-1-induced collective directional migration and angiogenic sprouting.Phosphorylation of VE-cadherin is modulated by haemodynamic forces and contributes to the regulation of vascular permeability in vivo.Role of Krev Interaction Trapped-1 in Prostacyclin-Induced Protection against Lung Vascular Permeability Induced by Excessive Mechanical Forces and Thrombin Receptor Activating Peptide 6.Apicobasal polarity of brain endothelial cells.
P2860
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P2860
CCM1 regulates vascular-lumen organization by inducing endothelial polarity
description
2010 nî lūn-bûn
@nan
2010 թուականի Ապրիլին հրատարակուած գիտական յօդուած
@hyw
2010 թվականի ապրիլին հրատարակված գիտական հոդված
@hy
2010年の論文
@ja
2010年論文
@yue
2010年論文
@zh-hant
2010年論文
@zh-hk
2010年論文
@zh-mo
2010年論文
@zh-tw
2010年论文
@wuu
name
CCM1 regulates vascular-lumen organization by inducing endothelial polarity
@ast
CCM1 regulates vascular-lumen organization by inducing endothelial polarity
@en
CCM1 regulates vascular-lumen organization by inducing endothelial polarity
@nl
type
label
CCM1 regulates vascular-lumen organization by inducing endothelial polarity
@ast
CCM1 regulates vascular-lumen organization by inducing endothelial polarity
@en
CCM1 regulates vascular-lumen organization by inducing endothelial polarity
@nl
prefLabel
CCM1 regulates vascular-lumen organization by inducing endothelial polarity
@ast
CCM1 regulates vascular-lumen organization by inducing endothelial polarity
@en
CCM1 regulates vascular-lumen organization by inducing endothelial polarity
@nl
P2093
P50
P921
P3181
P356
P1476
CCM1 regulates vascular-lumen organization by inducing endothelial polarity
@en
P2093
Fabrizio Orsenigo
Francoise Chapon
Luigi Maddaluno
Noemi Rudini
P304
P3181
P356
10.1242/JCS.059329
P407
P577
2010-04-01T00:00:00Z