Disruption of platelet-derived growth factor-dependent phosphatidylinositol 3-kinase and phospholipase Cγ 1 activity abolishes vascular smooth muscle cell proliferation and migration and attenuates neointima formation in vivo

Disruption of platelet-derived growth factor-dependent phosphatidylinositol 3-kinase and phospholipase Cγ 1 activity abolishes vascular smooth muscle cell proliferation and migration and attenuates neointima formation in vivo

Item
http://www.wikidata.org/entity/Q24306656
Myristoylated Alanine-Rich Protein Kinase Substrate (MARCKS) Regulates Small GTPase Rac1 and Cdc42 Activity and Is a Critical Mediator of Vascular Smooth Muscle Cell Migration in Intimal Hyperplasia Formation.Smooth Muscle Cell-targeted RNA Aptamer Inhibits Neointimal Formation.MicroRNA-182 prevents vascular smooth muscle cell dedifferentiation via FGF9/PDGFRβ signaling.Imatinib mesylate for the treatment of pulmonary arterial hypertension.Low-density lipoprotein receptor-related protein-1: role in the regulation of vascular integrity.Genetic Ablation of PDGF-Dependent Signaling Pathways Abolishes Vascular Remodeling and Experimental Pulmonary HypertensionPhospholipase Cγ1 Mediates Intima Formation Through Akt-Notch1 Signaling Independent of the Phospholipase Activity.Cinnamon and its Components Suppress Vascular Smooth Muscle Cell Proliferation by Up-Regulating Cyclin-Dependent Kinase Inhibitors.Inhibition of protein tyrosine phosphatases enhances cerebral collateral growth in rats.Kv1.3 channels modulate human vascular smooth muscle cells proliferation independently of mTOR signaling pathway.Antiproliferative Activity of Hinokitiol, a Tropolone Derivative, Is Mediated via the Inductions of p-JNK and p-PLCγ1 Signaling in PDGF-BB-Stimulated Vascular Smooth Muscle Cells.Antiplatelet and anticoagulant for prevention of reocclusion in patients with atrial fibrillation undergoing endovascular treatment for low extremity ischemia.FGF9/FGFR2 increase cell proliferation by activating ERK1/2, Rb/E2F1, and cell cycle pathways in mouse Leydig tumor cellsPI3Kγ promotes vascular smooth muscle cell phenotypic modulation and transplant arteriosclerosis via a SOX9-dependent mechanism
P2860

Disruption of platelet-derived growth factor-dependent phosphatidylinositol 3-kinase and phospholipase Cγ 1 activity abolishes vascular smooth muscle cell proliferation and migration and attenuates neointima formation in vivo

Item
http://www.wikidata.org/entity/Q24306656
description
2011 nî lūn-bûn
@nan
2011 թուականի Յունիսին հրատարակուած գիտական յօդուած
@hyw
2011 թվականի հունիսին հրատարակված գիտական հոդված
@hy
2011年の論文
@ja
2011年論文
@yue
2011年論文
@zh-hant
2011年論文
@zh-hk
2011年論文
@zh-mo
2011年論文
@zh-tw
2011年论文
@wuu
name
Disruption of platelet-derived ...... es neointima formation in vivo
@ast
Disruption of platelet-derived ...... es neointima formation in vivo
@en
Disruption of platelet-derived ...... es neointima formation in vivo
@en-gb
Disruption of platelet-derived ...... es neointima formation in vivo
@nl
type
label
Disruption of platelet-derived ...... es neointima formation in vivo
@ast
Disruption of platelet-derived ...... es neointima formation in vivo
@en
Disruption of platelet-derived ...... es neointima formation in vivo
@en-gb
Disruption of platelet-derived ...... es neointima formation in vivo
@nl
altLabel
Disruption of Platelet-Derived ...... es Neointima Formation In Vivo
@en
prefLabel
Disruption of platelet-derived ...... es neointima formation in vivo
@ast
Disruption of platelet-derived ...... es neointima formation in vivo
@en
Disruption of platelet-derived ...... es neointima formation in vivo
@en-gb
Disruption of platelet-derived ...... es neointima formation in vivo
@nl
P1433
P1476
P2093
P2860
P304
P31
P356
P407
P433
P478
P50
P577
P5875
P698
P921
P932
P356
P1433
P1476
Disruption of platelet-derived ...... es neointima formation in vivo
@en
P2093
P2860
P304
P31
P356
10.1016/J.JACC.2011.02.037
P407
P433
P478
P50
P577
P5875
P698
P921
P932