Atlastin GTPases are required for Golgi apparatus and ER morphogenesis.
about
MITD1 is recruited to midbodies by ESCRT-III and participates in cytokinesisHereditary spastic paraplegia proteins REEP1, spastin, and atlastin-1 coordinate microtubule interactions with the tubular ER networkRab10 GTPase regulates ER dynamics and morphology.Structural basis for conformational switching and GTP loading of the large G protein atlastinSpastin couples microtubule severing to membrane traffic in completion of cytokinesis and secretionProtrudin binds atlastins and endoplasmic reticulum-shaping proteins and regulates network formationScyl1, mutated in a recessive form of spinocerebellar neurodegeneration, regulates COPI-mediated retrograde trafficA class of dynamin-like GTPases involved in the generation of the tubular ER networkMembrane-shaping disorders: a common pathway in axon degenerationStructural basis for the nucleotide-dependent dimerization of the large G protein atlastin-1/SPG3AStructures of the atlastin GTPase provide insight into homotypic fusion of endoplasmic reticulum membranesDelving into the complexity of hereditary spastic paraplegias: how unexpected phenotypes and inheritance modes are revolutionizing their nosologyExome sequencing links corticospinal motor neuron disease to common neurodegenerative disordersA combined approach of quantitative interaction proteomics and live-cell imaging reveals a regulatory role for endoplasmic reticulum (ER) reticulon homology proteins in peroxisome biogenesisSpastin, atlastin, and ER relocalization are involved in axon but not dendrite regeneration.Hereditary spastic paraplegia: clinico-pathologic features and emerging molecular mechanisms.Further assembly required: construction and dynamics of the endoplasmic reticulum network.LC-MS/MS analysis of differentially expressed glioblastoma membrane proteome reveals altered calcium signaling and other protein groups of regulatory functions.Zebrafish atlastin controls motility and spinal motor axon architecture via inhibition of the BMP pathway.Targeted high-throughput sequencing identifies mutations in atlastin-1 as a cause of hereditary sensory neuropathy type I.Emerging themes of ER organization in the development and maintenance of axons.A lethal de novo mutation in the middle domain of the dynamin-related GTPase Drp1 impairs higher order assembly and mitochondrial division.Pharmacologic rescue of axon growth defects in a human iPSC model of hereditary spastic paraplegia SPG3A.The effect of HSP-causing mutations in SPG3A and NIPA1 on the assembly, trafficking, and interaction between atlastin-1 and NIPA1ERMO3/MVP1/GOLD36 is involved in a cell type-specific mechanism for maintaining ER morphology in Arabidopsis thalianaNeuropathy target esterase is required for adult vertebrate axon maintenanceFusing a lasting relationship between ER tubules.Balancing ER dynamics: shaping, bending, severing, and mending membranesUpregulation of the zebrafish Nogo-A homologue, Rtn4b, in retinal ganglion cells is functionally involved in axon regenerationGTP-dependent packing of a three-helix bundle is required for atlastin-mediated fusion.Human atlastin GTPases mediate differentiated fusion of endoplasmic reticulum membranes.Cis and trans interactions between atlastin molecules during membrane fusionER network formation and membrane fusion by atlastin1/SPG3A disease variants.An intramolecular salt bridge drives the soluble domain of GTP-bound atlastin into the postfusion conformationCompartmentalization of membrane trafficking, glucose transport, glycolysis, actin, tubulin and the proteasome in the cytoplasmic droplet/Hermes body of epididymal sperm.Nongenomic STAT5-dependent effects on Golgi apparatus and endoplasmic reticulum structure and functionSNAREs support atlastin-mediated homotypic ER fusion in Saccharomyces cerevisiaeISG15 counteracts Listeria monocytogenes infection.Lipid interaction of the C terminus and association of the transmembrane segments facilitate atlastin-mediated homotypic endoplasmic reticulum fusion.Weaving the web of ER tubules
P2860
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P248
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P2860
Atlastin GTPases are required for Golgi apparatus and ER morphogenesis.
description
2008 nî lūn-bûn
@nan
2008 թուականի Փետրուարին հրատարակուած գիտական յօդուած
@hyw
2008 թվականի փետրվարին հրատարակված գիտական հոդված
@hy
2008年の論文
@ja
2008年論文
@yue
2008年論文
@zh-hant
2008年論文
@zh-hk
2008年論文
@zh-mo
2008年論文
@zh-tw
2008年论文
@wuu
name
Atlastin GTPases are required for Golgi apparatus and ER morphogenesis
@nl
Atlastin GTPases are required for Golgi apparatus and ER morphogenesis.
@ast
Atlastin GTPases are required for Golgi apparatus and ER morphogenesis.
@en
Atlastin GTPases are required for Golgi apparatus and ER morphogenesis.
@en-gb
type
label
Atlastin GTPases are required for Golgi apparatus and ER morphogenesis
@nl
Atlastin GTPases are required for Golgi apparatus and ER morphogenesis.
@ast
Atlastin GTPases are required for Golgi apparatus and ER morphogenesis.
@en
Atlastin GTPases are required for Golgi apparatus and ER morphogenesis.
@en-gb
prefLabel
Atlastin GTPases are required for Golgi apparatus and ER morphogenesis
@nl
Atlastin GTPases are required for Golgi apparatus and ER morphogenesis.
@ast
Atlastin GTPases are required for Golgi apparatus and ER morphogenesis.
@en
Atlastin GTPases are required for Golgi apparatus and ER morphogenesis.
@en-gb
P2093
P2860
P921
P3181
P356
P1476
Atlastin GTPases are required for Golgi apparatus and ER morphogenesis.
@en
P2093
Craig Blackstone
Cynthia Soderblom
Julia Stadler
Neggy Rismanchi
Peng-Peng Zhu
P2860
P304
P3181
P356
10.1093/HMG/DDN046
P407
P577
2008-02-12T00:00:00Z