Inhibition of glioma cell growth and tumorigenic potential by CCN3 (NOV) - sprookjes - yvandenbroek - TriplyDB

Inhibition of glioma cell growth and tumorigenic potential by CCN3 (NOV)

Inhibition of glioma cell growth and tumorigenic potential by CCN3 (NOV)

http://www.wikidata.org/entity/Q24535398

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The nephroblastoma overexpressed gene (NOV/ccn3) protein associates with Notch1 extracellular domain and inhibits myoblast differentiation via Notch signaling pathway CCN3 (NOV) is a novel angiogenic regulator of the CCN protein family Integrin-dependent functions of the angiogenic inducer NOV (CCN3): implication in wound healing CCN proteins: A centralized communication network CCN3 impairs osteoblast and stimulates osteoclast differentiation to favor breast cancer metastasis to bone Potential cellular conformations of the CCN3(NOV) protein A structural approach to the role of CCN (CYR61/CTGF/NOV) proteins in tumourigenesis Inhibition of CCN6 (WISP3) expression promotes neoplastic progression and enhances the effects of insulin-like growth factor-1 on breast epithelial cells.Gap junctions Taking aim at the extracellular matrix: CCN proteins as emerging therapeutic targets Matricellular protein CCN3 (NOV) regulates actin cytoskeleton reorganization.A role for CCN3 (NOV) in calcium signalling.Recombinant expression, purification, and functional characterisation of connective tissue growth factor and nephroblastoma-overexpressed protein The CCN proteins: important signaling mediators in stem cell differentiation and tumorigenesis.Overexpression of WISP-1 down-regulated motility and invasion of lung cancer cells through inhibition of Rac activation.The gene encoding the hematopoietic stem cell regulator CCN3/NOV is under direct cytokine control through the transcription factors STAT5A/B.Intercellular redistribution of cAMP underlies selective suppression of cancer cell growth by connexin26.The CCN3 (NOV) cell growth regulator: a new tool for molecular medicine.CONNEXIN 43 AND BONE: NOT JUST A GAP JUNCTION PROTEIN.NOV story: the way to CCN3.Glucose metabolism and proliferation in glia: role of astrocytic gap junctions.Matricellular proteins in drug delivery: Therapeutic targets, active agents, and therapeutic localization Huaier restrains proliferative and invasive potential of human hepatoma SKHEP-1 cells partially through decreased Lamin B1 and elevated NOV.CCN family of proteins: critical modulators of the tumor cell microenvironment.Emerging role of CCN family proteins in tumorigenesis and cancer metastasis (Review).Nephroblastoma overexpressed (NOV/CCN3) gene: a paired-domain-specific PAX3-FKHR transcription target that promotes survival and motility in alveolar rhabdomyosarcoma cells.Human pancreatic cancer progression: an anarchy among CCN-siblings.CCN3 (NOV) regulates proliferation, adhesion, migration and invasion in clear cell renal cell carcinoma CCN3/NOV small interfering RNA enhances fibrogenic gene expression in primary hepatic stellate cells and cirrhotic fat storing cell line CFSC.Downregulation of CCN3 expression as a potential mechanism for melanoma progression.The expression of novH in adrenocortical cells is down-regulated by TGFbeta 1 through c-Jun in a Smad-independent manner.CCN3: Doctor Jekyll and Mister Hyde.Domain-specific CCN3 antibodies as unique tools for structural and functional studies.The matricellular protein CCN3 regulates NOTCH1 signalling in chronic myeloid leukaemia.Matricellular proteins produced by melanocytes and melanomas: in search for functions.Temporal and spatial expression of CCN3 during retina development.Gap junction protein Connexin-43 is a direct transcriptional regulator of N-cadherin in vivo

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Inhibition of glioma cell growth and tumorigenic potential by CCN3 (NOV)

http://www.wikidata.org/entity/Q24535398

description

2001 nî lūn-bûn

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2001 թուականի Հոկտեմբերին հրատարակուած գիտական յօդուած

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2001 թվականի հոտեմբերին հրատարակված գիտական հոդված

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2001年の論文

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2001年学术文章

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2001年学术文章

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2001年学术文章

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2001年学术文章

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2001年学术文章

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2001年學術文章

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name

Inhibition of glioma cell growth and tumorigenic potential by CCN3 (NOV)

@ast

Inhibition of glioma cell growth and tumorigenic potential by CCN3 (NOV)

@en

Inhibition of glioma cell growth and tumorigenic potential by CCN3 (NOV)

@en-gb

Inhibition of glioma cell growth and tumorigenic potential by CCN3 (NOV)

@nl

Inhibition of glioma cell growth and tumorigenic potential by CCN3 (NOV)

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type

label

Inhibition of glioma cell growth and tumorigenic potential by CCN3 (NOV)

@ast

Inhibition of glioma cell growth and tumorigenic potential by CCN3 (NOV)

@en

Inhibition of glioma cell growth and tumorigenic potential by CCN3 (NOV)

@en-gb

Inhibition of glioma cell growth and tumorigenic potential by CCN3 (NOV)

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Inhibition of glioma cell growth and tumorigenic potential by CCN3 (NOV)

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prefLabel

Inhibition of glioma cell growth and tumorigenic potential by CCN3 (NOV)

@ast

Inhibition of glioma cell growth and tumorigenic potential by CCN3 (NOV)

@en

Inhibition of glioma cell growth and tumorigenic potential by CCN3 (NOV)

@en-gb

Inhibition of glioma cell growth and tumorigenic potential by CCN3 (NOV)

@nl

Inhibition of glioma cell growth and tumorigenic potential by CCN3 (NOV)

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Journal of Clinical Pathology

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Inhibition of glioma cell growth and tumorigenic potential by CCN3 (NOV)

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B Perbal

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C C Naus

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H Wang

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J Yu

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N Gupta

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R R Weichselbaum

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S Advani

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S Kyurkchiev

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T L McLeod

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P2860

WISP genes are members of the connective tissue growth factor family that are up-regulated in wnt-1-transformed cells and aberrantly expressed in human colon tumors

The C-terminal domain of the regulatory protein NOVH is sufficient to promote interaction with fibulin 1C: a clue for a role of NOVH in cell-adhesion signaling

The human growth factor-inducible immediate early gene, CYR61, maps to chromosome 1p

Fisp12/mouse connective tissue growth factor mediates endothelial cell adhesion and migration through integrin alphavbeta3, promotes endothelial cell survival, and induces angiogenesis in vivo

CYR61, a product of a growth factor-inducible immediate early gene, promotes angiogenesis and tumor growth

Physical mapping of human loci homologous to the chicken nov proto-oncogene

NOV (nephroblastoma overexpressed) and the CCN family of genes: structural and functional issues.

Nephroblastoma overexpressed gene (NOV) codes for a growth factor that induces protein tyrosine phosphorylation

Proviral rearrangements and overexpression of a new cellular gene (nov) in myeloblastosis-associated virus type 1-induced nephroblastomas

Connective tissue growth factor: a cysteine-rich mitogen secreted by human vascular endothelial cells is related to the SRC-induced immediate early gene product CEF-10.

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10.1136/MP.54.5.293

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54

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11577170

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