Demonstration of factor H-like protein 1 binding to Treponema denticola, a pathogen associated with periodontal disease in humans.
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Structure of Factor H-binding Protein B (FhbB) of the Periopathogen,Treponema denticolaComplement control protein factor H: the good, the bad, and the inadequate.Analysis of the complement sensitivity of oral treponemes and the potential influence of FH binding, FH cleavage and dentilisin activity on the pathogenesis of periodontal disease.Molecular analyses of the interaction of Borrelia hermsii FhbA with the complement regulatory proteins factor H and factor H-like protein 1Identification of the primary mechanism of complement evasion by the periodontal pathogen, Treponema denticola.Genome-wide relatedness of Treponema pedis, from gingiva and necrotic skin lesions of pigs, with the human oral pathogen Treponema denticola.Protease-dependent mechanisms of complement evasion by bacterial pathogens.The Treponema denticola FhbB Protein Is a Dominant Early Antigen That Elicits FhbB Variant-Specific Antibodies That Block Factor H Binding and Cleavage by Dentilisin.Sequence divergence in the Treponema denticola FhbB protein and its impact on factor H binding.A surface-exposed neuraminidase affects complement resistance and virulence of the oral spirochaete Treponema denticola.The complement system and its role in the pathogenesis of periodontitis: current concepts.The Cross-Talk between Spirochetal Lipoproteins and Immunity.Utilizing complement evasion strategies to design complement-based antibacterial immunotherapeutics: Lessons from the pathogenic Neisseriae.Immune evasion of leptospira species by acquisition of human complement regulator C4BP.Identification of an antiparallel coiled-coil/loop domain required for ligand binding by the Borrelia hermsii FhbA protein: additional evidence for the role of FhbA in the host-pathogen interaction.The opportunistic human pathogenic fungus Aspergillus fumigatus evades the host complement system.Selective binding of Borrelia burgdorferi OspE paralogs to factor H and serum proteins from diverse animals: possible expansion of the role of OspE in Lyme disease pathogenesis.Evidence that the BBA68 protein (BbCRASP-1) of the Lyme disease spirochetes does not contribute to factor H-mediated immune evasion in humans and other animals.Identification of the gene encoding the FhbB protein of Treponema denticola, a highly unique factor H-like protein 1 binding protein.Gene Regulation, Two Component Regulatory Systems, and Adaptive Responses in Treponema Denticola.Complement Immune Evasion by Spirochetes.
P2860
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P2860
Demonstration of factor H-like protein 1 binding to Treponema denticola, a pathogen associated with periodontal disease in humans.
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2005 nî lūn-bûn
@nan
2005 թուականի Նոյեմբերին հրատարակուած գիտական յօդուած
@hyw
2005 թվականի նոյեմբերին հրատարակված գիտական հոդված
@hy
2005年の論文
@ja
2005年論文
@yue
2005年論文
@zh-hant
2005年論文
@zh-hk
2005年論文
@zh-mo
2005年論文
@zh-tw
2005年论文
@wuu
name
Demonstration of factor H-like ...... periodontal disease in humans
@nl
Demonstration of factor H-like ...... periodontal disease in humans.
@ast
Demonstration of factor H-like ...... periodontal disease in humans.
@en
type
label
Demonstration of factor H-like ...... periodontal disease in humans
@nl
Demonstration of factor H-like ...... periodontal disease in humans.
@ast
Demonstration of factor H-like ...... periodontal disease in humans.
@en
prefLabel
Demonstration of factor H-like ...... periodontal disease in humans
@nl
Demonstration of factor H-like ...... periodontal disease in humans.
@ast
Demonstration of factor H-like ...... periodontal disease in humans.
@en
P2093
P2860
P1476
Demonstration of factor H-like ...... periodontal disease in humans
@en
P2093
John V McDowell
Justin Lankford
Lola Stamm
Tania Sadlon
P2860
P304
P356
10.1128/IAI.73.11.7126-7132.2005
P407
P577
2005-11-01T00:00:00Z