HIV-1 Tat potentiates TNF-induced NF-kappa B activation and cytotoxicity by altering the cellular redox state
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HIV-1 Tat protein enhances microtubule polymerizationThe HIV-1 viral protein R induces apoptosis via a direct effect on the mitochondrial permeability transition poreAmplification of the inflammatory cellular redox state by human immunodeficiency virus type 1-immunosuppressive tat and gp160 proteinsTat protein of human immunodeficiency virus type 1 induces interleukin-10 in human peripheral blood monocytes: implication of protein kinase C-dependent pathway.Acetylation of histones and transcription-related factorsTransdominant mutants of I kappa B alpha block Tat-tumor necrosis factor synergistic activation of human immunodeficiency virus type 1 gene expression and virus multiplicationActivation of transcription factor NF-kappaB by the Tat protein of human immunodeficiency virus type 1A Tat-induced auto-up-regulatory loop for superactivation of the human immunodeficiency virus type 1 promoterModulation of macrophage functions by sheeppox virus provides clues to understand interaction of the virus with host immune system.Involvement of Reactive Oxygen Species, Glutathione Metabolism, and Lipid Peroxidation in the Cf-Gene-Dependent Defense Response of Tomato Cotyledons Induced by Race-Specific Elicitors of Cladosporium fulvumSynergistic neurotoxicity of opioids and human immunodeficiency virus-1 Tat protein in striatal neurons in vitroThe roles of HIV-1 proteins and antiretroviral drug therapy in HIV-1-associated endothelial dysfunctionSpecific increase in MDR1 mediated drug-efflux in human brain endothelial cells following co-exposure to HIV-1 and saquinavirGlutathione deficiency is associated with impaired survival in HIV disease.HIV-1 Tat-mediated apoptosis in human blood-retinal barrier-associated cells.Acetylated Tat regulates human immunodeficiency virus type 1 splicing through its interaction with the splicing regulator p32.Conserved cysteines of the human immunodeficiency virus type 1 protease are involved in regulation of polyprotein processing and viral maturation of immature virionsNuclear factor-kappa B family member RelB inhibits human immunodeficiency virus-1 Tat-induced tumor necrosis factor-alpha production.The initial step in human immunodeficiency virus type 1 GagProPol processing can be regulated by reversible oxidationInhibition of ascorbate peroxidase by salicylic acid and 2,6-dichloroisonicotinic acid, two inducers of plant defense responses.Vpr stimulates viral expression and induces cell killing in human immunodeficiency virus type 1-infected dividing Jurkat T cellsMethamphetamine abuse, HIV infection, and neurotoxicity.The vaccinia virus superoxide dismutase-like protein (A45R) is a virion component that is nonessential for virus replication.Modifications in host cell cytoskeleton structure and function mediated by intracellular HIV-1 Tat protein are greatly dependent on the second coding exonUsing death to one's advantage: HIV modulation of apoptosis.Chronic central nervous system expression of HIV-1 Tat leads to accelerated rarefaction of neocortical capillaries and loss of red blood cell velocity heterogeneity.Death of CD4(+) T-cell lines caused by human immunodeficiency virus type 1 does not depend on caspases or apoptosisHIV-1 pathogenicity and virion production are dependent on the metabolic phenotype of activated CD4+ T cellsActivation of Egr-1 expression in astrocytes by HIV-1 Tat: new insights into astrocyte-mediated Tat neurotoxicity.Measuring glutathione redox potential of HIV-1-infected macrophages.Neurodegenerative effects of recombinant HIV-1 Tat(1-86) are associated with inhibition of microtubule formation and oxidative stress-related reductions in microtubule-associated protein-2(a,b)Lipid Peroxidation Correlates with HIVmRNA in Serodiscordant Heterosexual HIVpartners of Nigerian Origin.HIV-1 Tat increases oxidant burden in the lungs of transgenic miceOn the selectivity of superoxide dismutase mimetics and its importance in pharmacological studies.Didehydro-cortistatin A inhibits HIV-1 Tat mediated neuroinflammation and prevents potentiation of cocaine reward in Tat transgenic miceNitric oxide and thiol redox regulation of Janus kinase activityEndotoxin-induced cytokine and chemokine expression in the HIV-1 transgenic rat.Human immunodeficiency virus type 1 Nef does not alter T-cell sensitivity to antigen-specific stimulation.HIV-1, reactive oxygen species, and vascular complicationsHIV-1 tat binds TAFII250 and represses TAFII250-dependent transcription of major histocompatibility class I genes.
P2860
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P2860
HIV-1 Tat potentiates TNF-induced NF-kappa B activation and cytotoxicity by altering the cellular redox state
description
1995 nî lūn-bûn
@nan
1995 թուականի Փետրուարին հրատարակուած գիտական յօդուած
@hyw
1995 թվականի փետրվարին հրատարակված գիտական հոդված
@hy
1995年の論文
@ja
1995年論文
@yue
1995年論文
@zh-hant
1995年論文
@zh-hk
1995年論文
@zh-mo
1995年論文
@zh-tw
1995年论文
@wuu
name
HIV-1 Tat potentiates TNF-indu ...... ering the cellular redox state
@ast
HIV-1 Tat potentiates TNF-indu ...... ering the cellular redox state
@en
HIV-1 Tat potentiates TNF-indu ...... ering the cellular redox state
@nl
type
label
HIV-1 Tat potentiates TNF-indu ...... ering the cellular redox state
@ast
HIV-1 Tat potentiates TNF-indu ...... ering the cellular redox state
@en
HIV-1 Tat potentiates TNF-indu ...... ering the cellular redox state
@nl
prefLabel
HIV-1 Tat potentiates TNF-indu ...... ering the cellular redox state
@ast
HIV-1 Tat potentiates TNF-indu ...... ering the cellular redox state
@en
HIV-1 Tat potentiates TNF-indu ...... ering the cellular redox state
@nl
P2093
P2860
P1433
P1476
HIV-1 Tat potentiates TNF-indu ...... ering the cellular redox state
@en
P2093
Krammer PH
Schulze-Osthoff K
Shatrov VA
Westendorp MO
P2860
P304
P356
10.1002/J.1460-2075.1995.TB07030.X
P407
P577
1995-02-01T00:00:00Z