Role of nuclear receptor corepressor RIP140 in metabolic syndrome
about
Transcription factor regulation in adipogenesisEstrogen-related receptor β deficiency alters body composition and response to restraint stress.TRPV1 activation counters diet-induced obesity through sirtuin-1 activation and PRDM-16 deacetylation in brown adipose tissue.A systems biology analysis of brain microvascular endothelial cell lipotoxicityImmune regulation of metabolic homeostasis in health and disease.Pharmacological manipulation of transcription factor protein-protein interactions: opportunities and obstaclesProtein arginine methyltransferase 5 (Prmt5) promotes gene expression of peroxisome proliferator-activated receptor γ2 (PPARγ2) and its target genes during adipogenesis.Transgenic mouse models resistant to diet-induced metabolic disease: is energy balance the key?Transcriptional Regulation by Nuclear Corepressors and PGC-1α: Implications for Mitochondrial Quality Control and Insulin Sensitivity.SMRT-GPS2 corepressor pathway dysregulation coincides with obesity-linked adipocyte inflammation.Stabilization of the promoter nucleosomes in nucleosome-free regions by the yeast Cyc8-Tup1 corepressor.Integrative genomics of gene and metabolic regulation by estrogen receptors α and β, and their coregulators.RIP140, a Janus metabolic switch involved in defense functions.Role of microRNAs on the Regulation of Mitochondrial Biogenesis and Insulin Signaling in Skeletal Muscle.Cardiac nuclear receptors: architects of mitochondrial structure and function.The Engrailed-1 Gene Stimulates Brown Adipogenesis.Receptor-interacting Protein 140 represses Sirtuin 3 to facilitate hypertrophy, mitochondrial dysfunction and energy metabolic dysfunction in cardiomyocytes.Eicosapentaenoic Acid Potentiates Brown Thermogenesis through FFAR4-dependent Up-regulation of miR-30b and miR-378.Complex regulation of LCoR signaling in breast cancer cells.Regulation of Nuclear Receptor Interacting Protein 1 (NRIP1) Gene Expression in Response to Weight Loss and Exercise in Humans.Deletion of Nrip1 extends female mice longevity, increases autophagy, and delays cell senescence.Downregulation of RIP140 in triple-negative breast cancer inhibits the growth and proliferation of cancer cells.
P2860
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P2860
Role of nuclear receptor corepressor RIP140 in metabolic syndrome
description
2011 nî lūn-bûn
@nan
2011 թուականի Օգոստոսին հրատարակուած գիտական յօդուած
@hyw
2011 թվականի օգոստոսին հրատարակված գիտական հոդված
@hy
2011年の論文
@ja
2011年論文
@yue
2011年論文
@zh-hant
2011年論文
@zh-hk
2011年論文
@zh-mo
2011年論文
@zh-tw
2011年论文
@wuu
name
Role of nuclear receptor corepressor RIP140 in metabolic syndrome
@ast
Role of nuclear receptor corepressor RIP140 in metabolic syndrome
@en
Role of nuclear receptor corepressor RIP140 in metabolic syndrome
@nl
type
label
Role of nuclear receptor corepressor RIP140 in metabolic syndrome
@ast
Role of nuclear receptor corepressor RIP140 in metabolic syndrome
@en
Role of nuclear receptor corepressor RIP140 in metabolic syndrome
@nl
prefLabel
Role of nuclear receptor corepressor RIP140 in metabolic syndrome
@ast
Role of nuclear receptor corepressor RIP140 in metabolic syndrome
@en
Role of nuclear receptor corepressor RIP140 in metabolic syndrome
@nl
P2093
P2860
P921
P3181
P1476
Role of nuclear receptor corepressor RIP140 in metabolic syndrome
@en
P2093
Malcolm G Parker
Marius C Jones
Meritxell Rosell
P2860
P304
P3181
P356
10.1016/J.BBADIS.2010.12.016
P407
P5008
P577
2011-08-01T00:00:00Z