Inositol phosphatase SHIP1 is a primary target of miR-155 - sprookjes - yvandenbroek - TriplyDB

Inositol phosphatase SHIP1 is a primary target of miR-155

Inositol phosphatase SHIP1 is a primary target of miR-155

http://www.wikidata.org/entity/Q24648984

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Regulation of miRNA transcription in macrophages in response to Candida albicans MiR-155 induction by F. novicida but not the virulent F. tularensis results in SHIP down-regulation and enhanced pro-inflammatory cytokine response MicroRNAs enriched in hematopoietic stem cells differentially regulate long-term hematopoietic output Nanoparticle-based therapy in an in vivo microRNA-155 (miR-155)-dependent mouse model of lymphoma Efficient inhibition of miR-155 function in vivo by peptide nucleic acids MicroRNA-155 regulates cell survival, growth, and chemosensitivity by targeting FOXO3a in breast cancer Virally induced cellular microRNA miR-155 plays a key role in B-cell immortalization by Epstein-Barr virus IL-10 inhibits miR-155 induction by toll-like receptors MicroRNAs in mouse models of lymphoid malignancies MicroRNA-155 promotes autoimmune inflammation by enhancing inflammatory T cell development Targeting of SMAD5 links microRNA-155 to the TGF-beta pathway and lymphomagenesis Oncogenic IRFs provide a survival advantage for Epstein-Barr virus- or human T-cell leukemia virus type 1-transformed cells through induction of BIC expression The kinase Akt1 controls macrophage response to lipopolysaccharide by regulating microRNAs Interferon regulatory factor 3 inhibits astrocyte inflammatory gene expression through suppression of the proinflammatory miR-155 and miR-155*MicroRNAs in immune regulation--opportunities for cancer immunotherapy Inflammatory cytokines regulate microRNA-155 expression in human retinal pigment epithelial cells by activating JAK/STAT pathway Antagonism of microRNA-126 suppresses the effector function of TH2 cells and the development of allergic airways disease Reticuloendotheliosis virus strain T induces miR-155, which targets JARID2 and promotes cell survival Src homology 2 domain-containing inositol-5-phosphatase and CCAAT enhancer-binding protein beta are targeted by miR-155 in B cells of Emicro-MiR-155 transgenic mice Silencing of microRNA-155 in mice during acute inflammatory response leads to derepression of c/ebp Beta and down-regulation of G-CSF miR-155 as a Biomarker in B-Cell Malignancies Targeting microRNAs as key modulators of tumor immune response Expression and Function of miR-155 in Diseases of the Gastrointestinal Tract MicroRNA-155 and Its Role in Malignant Hematopoiesis Epigenetic: A missing paradigm in cellular and molecular pathways of sulfur mustard lung: a prospective and comparative study Regulation of Bim in Health and Disease MicroRNAs and the resolution phase of inflammation in macrophages Milk: an exosomal microRNA transmitter promoting thymic regulatory T cell maturation preventing the development of atopy?Natural killer cell regulation by microRNAs in health and disease Functional regulation of monocyte-derived dendritic cells by microRNAs Progress and challenge of microRNA research in immunity MicroRNAs: new regulators of Toll-like receptor signalling pathways Phosphoinositides: tiny lipids with giant impact on cell regulation MicroRNA-regulated pathways in hematological malignancies: how to avoid cells playing out of tune Role of microRNA deregulation in the pathogenesis of diffuse large B-cell lymphoma (DLBCL)Immune mechanisms in cerebral ischemic tolerance miRNA Deregulation in Cancer Cells and the Tumor Microenvironment miRNA dysregulation in cancer: towards a mechanistic understanding Antagonistic Interplay between MicroRNA-155 and IL-10 during Lyme Carditis and Arthritis The prognostic and functional role of microRNAs in acute myeloid leukemia

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P2860

Inositol phosphatase SHIP1 is a primary target of miR-155

http://www.wikidata.org/entity/Q24648984

description

2009 nî lūn-bûn

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2009 թուականի Ապրիլին հրատարակուած գիտական յօդուած

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2009 թվականի ապրիլին հրատարակված գիտական հոդված

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2009年の論文

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2009年論文

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2009年論文

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2009年論文

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2009年論文

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2009年論文

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2009年论文

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name

Inositol phosphatase SHIP1 is a primary target of miR-155

@ast

Inositol phosphatase SHIP1 is a primary target of miR-155

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Inositol phosphatase SHIP1 is a primary target of miR-155

@nl

type

label

Inositol phosphatase SHIP1 is a primary target of miR-155

@ast

Inositol phosphatase SHIP1 is a primary target of miR-155

@en

Inositol phosphatase SHIP1 is a primary target of miR-155

@nl

prefLabel

Inositol phosphatase SHIP1 is a primary target of miR-155

@ast

Inositol phosphatase SHIP1 is a primary target of miR-155

@en

Inositol phosphatase SHIP1 is a primary target of miR-155

@nl

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PNAS.0902636106

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Proceedings of the National Academy of Sciences of the United States of America

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Inositol phosphatase SHIP1 is a primary target of miR-155

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P2093

Aadel A Chaudhuri

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David Baltimore

http://www.w3.org/2001/XMLSchema#string

Ryan M O'Connell

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P2860

Polycistronic RNA polymerase II expression vectors for RNA interference based on BIC/miR-155.

Accumulation of miR-155 and BIC RNA in human B cell lymphomas

Foxp3-dependent microRNA155 confers competitive fitness to regulatory T cells by targeting SOCS1 protein

Targeted deletion reveals essential and overlapping functions of the miR-17 through 92 family of miRNA clusters

MicroRNA-155 suppresses activation-induced cytidine deaminase-mediated Myc-Igh translocation

bic, a novel gene activated by proviral insertions in avian leukosis virus-induced lymphomas, is likely to function through its noncoding RNA

Sustained expression of microRNA-155 in hematopoietic stem cells causes a myeloproliferative disorder

MicroRNA-155 is an Epstein-Barr virus-induced gene that modulates Epstein-Barr virus-regulated gene expression pathways

Lymphoproliferative disease and autoimmunity in mice with increased miR-17-92 expression in lymphocytes

Distinctive microRNA signature of acute myeloid leukemia bearing cytoplasmic mutated nucleophosmin

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scholarly article

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10.1073/PNAS.0902636106

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106

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19359473

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2678424

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