Telomere maintenance and dysfunction predict recurrence in paediatric ependymoma - sprookjes - yvandenbroek - TriplyDB

Telomere maintenance and dysfunction predict recurrence in paediatric ependymoma

Telomere maintenance and dysfunction predict recurrence in paediatric ependymoma

http://www.wikidata.org/entity/Q24652958

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Identification of microRNAs as potential prognostic markers in ependymoma MicroRNA Regulation of Brain Tumour Initiating Cells in Central Nervous System Tumours Molecular genetics of ependymoma Children's Oncology Group's 2013 blueprint for research: central nervous system tumors.Intratumoral diversity of telomere length in individual neuroblastoma tumors.Predictors of outcome in an AIEOP series of childhood ependymomas: a multifactorial analysis Chromosome 1q gain and tenascin-C expression are candidate markers to define different risk groups in pediatric posterior fossa ependymoma Histology and molecular pathology of pediatric brain tumors.Current and evolving knowledge of prognostic factors for pediatric ependymomas.Ependymoma in children: molecular considerations and therapeutic insights.Telomerase activation in posterior fossa group A ependymomas is associated with dismal prognosis and chromosome 1q gain.A combination of the telomerase inhibitor, BIBR1532, and paclitaxel synergistically inhibit cell proliferation in breast cancer cell lines.Direct short-term cytotoxic effects of BIBR 1532 on acute promyelocytic leukemia cells through induction of p21 coupled with downregulation of c-Myc and hTERT transcription.Telomere dysfunction and chromothripsis.

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P2860

Telomere maintenance and dysfunction predict recurrence in paediatric ependymoma

http://www.wikidata.org/entity/Q24652958

description

2008 nî lūn-bûn

@nan

2008 թուականի Հոկտեմբերին հրատարակուած գիտական յօդուած

@hyw

2008 թվականի հոտեմբերին հրատարակված գիտական հոդված

@hy

2008年の論文

@ja

2008年論文

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2008年論文

@zh-hant

2008年論文

@zh-hk

2008年論文

@zh-mo

2008年論文

@zh-tw

2008年论文

@wuu

name

Telomere maintenance and dysfunction predict recurrence in paediatric ependymoma

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Telomere maintenance and dysfunction predict recurrence in paediatric ependymoma

@en

Telomere maintenance and dysfunction predict recurrence in paediatric ependymoma

@nl

type

label

Telomere maintenance and dysfunction predict recurrence in paediatric ependymoma

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Telomere maintenance and dysfunction predict recurrence in paediatric ependymoma

@en

Telomere maintenance and dysfunction predict recurrence in paediatric ependymoma

@nl

prefLabel

Telomere maintenance and dysfunction predict recurrence in paediatric ependymoma

@ast

Telomere maintenance and dysfunction predict recurrence in paediatric ependymoma

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Telomere maintenance and dysfunction predict recurrence in paediatric ependymoma

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British Journal of Cancer

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Telomere maintenance and dysfunction predict recurrence in paediatric ependymoma

@en

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C Hawkins

http://www.w3.org/2001/XMLSchema#string

D Malkin

http://www.w3.org/2001/XMLSchema#string

E Bouffet

http://www.w3.org/2001/XMLSchema#string

J Ma

http://www.w3.org/2001/XMLSchema#string

J Rutka

http://www.w3.org/2001/XMLSchema#string

M Shago

http://www.w3.org/2001/XMLSchema#string

N Alon

http://www.w3.org/2001/XMLSchema#string

U Bartels

http://www.w3.org/2001/XMLSchema#string

U Tabori

http://www.w3.org/2001/XMLSchema#string

V Wong

http://www.w3.org/2001/XMLSchema#string

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The Hallmarks of Cancer

DNA damage foci at dysfunctional telomeres

Tumor stem cells derived from glioblastomas cultured in bFGF and EGF more closely mirror the phenotype and genotype of primary tumors than do serum-cultured cell lines

A DNA damage checkpoint response in telomere-initiated senescence

Telomerase inhibitors.

Infrequency of p53 gene mutations in ependymomas.

Tissue microarray profiling of cancer specimens and cell lines: opportunities and limitations.

p53 Pathway dysfunction in primary childhood ependymomas.

Human telomere reverse transcriptase expression predicts progression and survival in pediatric intracranial ependymoma.

Telomerase in brain tumors.

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1129-35

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scholarly article

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3956190

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10.1038/SJ.BJC.6604652

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7

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99

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2008-10-07T00:00:00Z

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23261145

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1002243079

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18797459

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2567068

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