Bmi-1 dependence distinguishes neural stem cell self-renewal from progenitor proliferation - sprookjes - yvandenbroek - TriplyDB

Bmi-1 dependence distinguishes neural stem cell self-renewal from progenitor proliferation

Bmi-1 dependence distinguishes neural stem cell self-renewal from progenitor proliferation

http://www.wikidata.org/entity/Q24652976

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Bypass of senescence by the polycomb group protein CBX8 through direct binding to the INK4A-ARF locus Regeneration of glomerular podocytes by human renal progenitors Identification of a polymorphism in the RING finger of human Bmi-1 that causes its degradation by the ubiquitin-proteasome system PTEN inhibits BMI1 function independently of its phosphatase activity MAPKAP kinase MK2 maintains self-renewal capacity of haematopoietic stem cells Several distinct polycomb complexes regulate and co-localize on the INK4a tumor suppressor locus Ubiquitin-specific proteases 7 and 11 modulate Polycomb regulation of the INK4a tumour suppressor Microarray analysis identifies a death-from-cancer signature predicting therapy failure in patients with multiple types of cancer Stable X chromosome inactivation involves the PRC1 Polycomb complex and requires histone MACROH2A1 and the CULLIN3/SPOP ubiquitin E3 ligase Mammary stem cells, self-renewal pathways, and carcinogenesis Bmi-1 promotes neural stem cell self-renewal and neural development but not mouse growth and survival by repressing the p16Ink4a and p19Arf senescence pathways.Geminin regulates neuronal differentiation by antagonizing Brg1 activity Human stem/progenitor cells from bone marrow promote neurogenesis of endogenous neural stem cells in the hippocampus of mice.Epigenetic choreographers of neurogenesis in the adult mammalian brain EZH1 and EZH2 cogovern histone H3K27 trimethylation and are essential for hair follicle homeostasis and wound repair Bmi1, stem cells, and senescence regulation PLAGL2 regulates Wnt signaling to impede differentiation in neural stem cells and gliomas Epigenetic regulation of neurogenesis in the adult mammalian brain Adult neurogenesis in the mammalian brain: significant answers and significant questions DHODH modulates transcriptional elongation in the neural crest and melanoma Cellular senescence and tumor suppressor gene p16 MiR-15a and MiR-16 control Bmi-1 expression in ovarian cancer Signaling mechanisms regulating adult neural stem cells and neurogenesis The polycomb group gene Bmi1 regulates antioxidant defenses in neurons by repressing p53 pro-oxidant activity Hmga2 promotes neural stem cell self-renewal in young but not old mice by reducing p16Ink4a and p19Arf Expression NSPc1 is a cell growth regulator that acts as a transcriptional repressor of p21Waf1/Cip1 via the RARE element Contribution of polycomb homologues Bmi-1 and Mel-18 to medulloblastoma pathogenesis The Polycomb group proteins bind throughout the INK4A-ARF locus and are disassociated in senescent cells Mel-18, a polycomb group protein, regulates cell proliferation and senescence via transcriptional repression of Bmi-1 and c-Myc oncoproteins Is Melanoma a stem cell tumor? Identification of neurogenic proteins in trans-differentiated cells Genetic alterations and in vivo tumorigenicity of neurospheres derived from an adult glioblastoma Absence of mutations in the coding sequence of the potential tumor suppressor 3pK in metastatic melanoma Post-Translational Modifications of Histones in Vertebrate Neurogenesis Lingual Epithelial Stem Cells and Organoid Culture of Them The Perivascular Niche and Self-Renewal of Stem Cells A Novel Aspect of Tumorigenesis-BMI1 Functions in Regulating DNA Damage Response Common stemness regulators of embryonic and cancer stem cells Primo-Vascular System as Presented by Bong Han Kim.Polycomb Group (PcG) Proteins and Human Cancers: Multifaceted Functions and Therapeutic Implications PRC2 during vertebrate organogenesis: a complex in transition

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Bmi-1 dependence distinguishes neural stem cell self-renewal from progenitor proliferation

http://www.wikidata.org/entity/Q24652976

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2003 nî lūn-bûn

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2003 թուականի Հոկտեմբերին հրատարակուած գիտական յօդուած

@hyw

2003 թվականի հոտեմբերին հրատարակված գիտական հոդված

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2003年の論文

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2003年論文

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2003年論文

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2003年論文

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2003年論文

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2003年論文

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Bmi-1 dependence distinguishes ...... from progenitor proliferation

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Bmi-1 dependence distinguishes ...... from progenitor proliferation

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Bmi-1 dependence distinguishes ...... from progenitor proliferation

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Bmi-1 dependence distinguishes ...... from progenitor proliferation

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Bmi-1 dependence distinguishes ...... from progenitor proliferation

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Bmi-1 dependence distinguishes ...... from progenitor proliferation

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Bmi-1 dependence distinguishes ...... from progenitor proliferation

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Bmi-1 dependence distinguishes ...... from progenitor proliferation

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Bmi-1 dependence distinguishes ...... from progenitor proliferation

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P2093

In-Kyung Park

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Michael F Clarke

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Toshihide Iwashita

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P2860

Transformation of axial skeleton due to overexpression of bmi-1 in transgenic mice

Alternative neural crest cell fates are instructively promoted by TGFbeta superfamily members

Control of the replicative life span of human fibroblasts by p16 and the polycomb protein Bmi-1

Neural crest stem cells persist in the adult gut but undergo changes in self-renewal, neuronal subtype potential, and factor responsiveness

Bmi-1 is required for maintenance of adult self-renewing haematopoietic stem cells

Tumor suppression by Ink4a-Arf: progress and puzzles

bmi-1 transgene induces lymphomas and collaborates with myc in tumorigenesis

Posterior transformation, neurological abnormalities, and severe hematopoietic defects in mice with a targeted deletion of the bmi-1 proto-oncogene

Mice doubly deficient for the Polycomb Group genes Mel18 and Bmi1 reveal synergy and requirement for maintenance but not initiation of Hox gene expression

Bmi-1 determines the proliferative capacity of normal and leukaemic stem cells

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scholarly article

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263727

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10.1038/NATURE02060

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6961

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425

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Sean J Morrison

Anna V Molofsky

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2003-10-30T00:00:00Z

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9042543

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1013519146

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14574365

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2614897

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